Platelet electrophoresis time measurement

The internal viscosity of platelets is greater than that of red blood cells. Increased platelet count, increased adhesion and aggregation, and increased releases all increase blood viscosity. Factors affecting platelet aggregation are the same as those affecting red blood cells. However, in low rheology, platelets are more likely to aggregate. Platelet adhesion, increased aggregation, increased whole blood viscosity, increased plasma composition and HCT, reduced platelet deformability, slowed blood flow velocity and abnormal flow patterns, and the formation of eddy currents are favorable conditions for the formation of thrombus. Vessel wall damage causes increased platelet adhesion and aggregation, changes in platelet number and morphology, and reduces blood fluidity. The blood flow is slowed or flowing through the curved, bifurcation, stenosis of the blood vessel, or the wall of the blood vessel is roughened by pathological damage. Platelets tend to adhere to the wall of the tube, aggregate and release procoagulant substances, so that blood coagulates and thrombus forms.

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