Myocardial infarction with mitral regurgitation

Myocardial infarction lesions involving the papillary muscles can produce varying degrees of mitral regurgitation. About 3% of people with coronary atherosclerotic heart disease have a mitral regurgitation after a cardiac catheterization. Coronary heart disease-induced mitral regurgitation can be caused by acute or chronic papillary muscle ischemia. During myocardial infarction, the papillary muscles can be completely broken within hours due to acute ischemic necrosis. Although there were no abnormalities in the chordae and leaflets, the mitral valve leaflets of the corresponding site lost the opening and closing function, and severe mitral regurgitation was present early after the infarction. In cases of acute myocardial infarction, about 0.4 to 5% of those who died of sudden severe mitral insufficiency due to papillary muscle rupture. Although myocardial infarction in some patients caused ischemic necrosis of the papillary muscles, but it did not completely break immediately, or due to long-term ischemia, the necrotic myocardial tissue was gradually replaced by fibrous tissue. Mitral valve insufficiency was present more than 2 months after myocardial embolization.

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