Renal atheroembolism

Renal Atherosclerosis Renal Atherosclerosis A clinical syndrome involves a rapid or slow decline in renal function based on the amount of atherosclerotic obstructed renal arteries. The incidence of renal embolism increases with age. The most important non-cardiogenic renal emboli is a ruptured aortic atheroma. Renal atherosclerosis can occur spontaneously during diffuse erosive atherosclerosis or secondary to vascular surgery, angioplasty, or angiography. Renal atheromas are typically non-expandable and irregular in shape, and are more likely to produce incomplete occlusion with secondary ischemic atrophy rather than renal infarction. Foreign body reactions often occur immediately, causing intimal hyperplasia, giant cell formation and hyperplasia, and further narrowing of the vessel lumen. Pathological reactions may contribute to a progressive decline in renal function often 3 to 8 weeks after atheroembolism.

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