Vasospasm

Introduction Vasospasm refers to an abnormal contraction state of an artery over a period of time due to external factors or its own factors. Patients with cerebral vasospasm will have repeated headaches, dizziness, memory loss, mood disorders, sleep disorders and other symptoms, should pay attention to reasonable diet, moderate exercise, smoking cessation and alcohol restriction and maintain psychological balance.

Cause

Arteriosclerotic plaques of the internal carotid artery or vertebral-basal artery system narrow the vascular lumen and cause eddy currents. When the eddy current accelerates, the blood vessel wall is stimulated to cause vasospasm and a transient ischemic attack occurs, and the symptoms disappear when the vortex decelerates. However, some scholars believe that due to the special nature of the cerebral vascular structure, it is not easy to occur. However, most scholars believe that vasospasm can undoubtedly occur in the internal carotid artery and the cerebral artery ring, cerebral angiography can be seen in the aortic stenosis; subarachnoid hemorrhage can cause extensive and focal cerebral vasospasm; brain surgery on the brain When the aorta is operated, the diameter of the artery is significantly thinned. Therefore, cerebral arterial spasm can also be caused by persistent hypertension, local injury or microparticle stimulation, and cause transient ischemic attack.

an examination

Related inspection

Brain CT examination, brain ultrasound examination, neurological examination

1, blood pressure check.

2. Neurological examination.

3, brain CT examination.

Differential diagnosis

Need to be differentiated from the following diseases:

First, focal epileptic seizures of various types of focal seizures have similarities with TIA, such as epileptic seizures or motor seizures are easily confused with TIA. Tension-free seizures are similar to those of a trip. It is more convenient to perform 24-hour EEG Holter monitoring. If there is focal epileptic discharge, it can be diagnosed as epilepsy. If there is no abnormality, it is considered as TIA. CT or MRI findings have focal non-infarct lesions in the brain and may also be considered epilepsy.

Second, Meniere's disease has a long duration of vertigo (up to 2-3 days), accompanied by tinnitus, hearing loss after multiple episodes, and no other signs of nervous system localization.

Third, before the syncope, there are many eyes black, dizziness and unstable standing, accompanied by pale, cold sweat, fine pulse and blood pressure drop, and transient disturbance of consciousness but quickly recovered after falling to the ground, and no nerve positioning Signs. More than an upright position occurs.

Fourth, migraine attacks in adolescence, often family history, episodes of unilateral headache, vomiting and other autonomic symptoms, less focal neurological loss, seizure time is also longer. Regardless of the factors, TIA should be considered as an important risk factor for complete stroke, especially in the short-term repeated authors. The disease can be relieved by itself, and treatment focuses on preventing recurrence.

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