Limb shortening

Introduction

Introduction Limb lingual deformity: congenital tibia lack of patient claudication, calf shortening. It can be seen that the humerus arch deformity, the foot valgus, the external hemorrhoids disappear, and accompanied by other limb shortening deformities.

Cause

Cause

(A) the cause of the disease In the early stage of the embryo, the limb was originally formed based on the loss of 8 weeks ago, causing deformity.

(two) pathogenesis

Some people think that the lack of the humerus secondary to the lesions of the limb muscles, the shortening of the sacral and calf triceps will enhance the stress of the tibia and the foot, often causing the calf arch and the foot valgus deformity.

Examine

an examination

Related inspection

CT examination of bone and joint and bone and joint of limbs and limbs

Minhang, the calf is short. It can be seen that the humerus arch deformity, the foot valgus, the external hemorrhoids disappear, and accompanied by other limb shortening deformities.

Coventry and Johnson divided the disease into three types:

Type I: The unilateral part is missing, the calf can be moderately shortened, and generally there is no disability.

Type II: The tibia is almost completely missing, the limb is extremely short, and the tibia is deformed in the middle 1/3 and the lower 1/3. The skin is dimpled, but has no adhesion to the arch end; the foot is drooping and everted; the ipsilateral femur is also shortened. Even if treated, the function is poor.

Type III: May be unilateral or bilateral, with other serious abnormalities, upper extremity or femoral deformities, and spina bifida. There are many such cases and the prognosis is poor.

Diagnosis can be established based on clinical presentation and X-ray films.

Diagnosis

Differential diagnosis

Limb growth and excessive thickening are one of the clinical manifestations of congenital arteriovenous fistula. Congenital arteriovenous fistula is caused by abnormal development of the embryonic primordium during the evolution process, resulting in abnormal traffic between arteries and veins. Abnormal posture of the limb or trunk: Dystonia is an involuntary movement. It is a group of symptoms of involuntary movement and abnormal position and posture of a part of the body caused by the uncoordinated and intermittent continuous contraction of the synergistic muscles of the body skeletal muscles and the antagonistic muscles.

Also known as Dystonia Syndrome. The cause may be unknown by central nervous system dysfunction (such as basal ganglia lesions), the former is called secondary dystonia, and the latter is called primary dystonia. Dystonia is characterized by an abnormal posture of the limb or trunk that can affect movement or cause a distorted, odd-asynchronous state.

Limb stiffness: stiffness and mobility disorders are common in rheumatism. Patients often feel joints, limbs, and waist when they start activities in the morning, or when they change to another posture after a certain posture in the body for a long time. The affected part has stiffness and pain.

Minhang, the calf is short. It can be seen that the humerus arch deformity, the foot valgus, the external hemorrhoids disappear, and accompanied by other limb shortening deformities.

Coventry and Johnson divided the disease into three types:

Type I: The unilateral part is missing, the calf can be moderately shortened, and generally there is no disability.

Type II: The tibia is almost completely missing, the limb is extremely short, and the tibia is deformed in the middle 1/3 and the lower 1/3. The skin is dimpled, but has no adhesion to the arch end; the foot is drooping and everted; the ipsilateral femur is also shortened. Even if treated, the function is poor.

Type III: May be unilateral or bilateral, with other serious abnormalities, upper extremity or femoral deformities, and spina bifida. There are many such cases and the prognosis is poor.

Diagnosis can be established based on clinical presentation and X-ray films.

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