Shock nerve involvement

Introduction Shock nerve involvement refers to the nerves being paralyzed by direct or indirect damage from external forces, and they can recover their symptoms from about one to three months.

Cause

The cause of shock nerve involvement

Clinically, it can be seen in direct nerve impact, nerve compression is relieved, and inflammation invades the nerve and causes neuroedema. The pathogenic mechanism is that the affected nerve does not get good blood supply and nutrition to restore nerve function, and some patients have poor function recovery. The longer the nerve ischemia time, the worse the recovery function.

an examination

Related inspection

Brain CT examination of urinary leucine leucine aminopeptidase

Shock neurological examination

Clinically more common in brain trauma, spinal cord compression, myelitis, peripheral neuritis, traumatic neuroedema. A severe disorder of arterial resistance regulation, loss of vascular tone, causing vasodilation, resulting in decreased peripheral vascular resistance and shock with reduced blood volume.

Differential diagnosis

Differential diagnosis of confusing symptoms of shock nerve involvement:

Pathological nerve damage: Neuropathic pain is usually caused by peripheral nerve injury. The nerve fibers in the damaged area can produce a large amount of abnormal electrical activity in the short term after the injury. Including chronic compression nerve injury and incomplete neurological injury models, it was found that in the early stages of nerve injury, abnormally blocking the aberrant electrical activity of the afferent nerve permanently prevented the model animals from producing pain sensitivity to thermal and mechanical stimuli.

Hypoxic ischemic nerve injury: MRI skull base thin layer scanning can even see nerve root swelling, hemorrhage, fracture; electrophysiological examination evoked potential can detect optic nerve and auditory nerve injury; EMG can measure facial nerve injury and judge prognosis .

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