Ulcers have a cauliflower-like appearance

Introduction More common in squamous epithelium, often occurs in the original squamous epithelium covering parts, such as skin, mouth, lips, cervix, vagina, esophagus, penis, etc., can also occur in squamous metaplasia of the site such as bronchi, gallbladder, In the renal pelvis and other places, although it is not covered by the squamous epithelium in normal, it can cause squamous cell carcinoma through squamous metaplasia. The naked eye is cauliflower-like and can be infiltrated into the deep layer. Skin ulcers are generally localized skin tissue defects caused by traumatic microbial infections, tumors, circulatory disorders and neurological dysfunction, immune dysfunction or congenital skin defects.

Cause

Skin ulcers are generally localized skin tissue defects caused by traumatic microbial infections, tumors, circulatory disorders and neurological dysfunction, immune dysfunction or congenital skin defects. Traumatic ulcers are often caused by physical and chemical factors acting directly on tissues. Microbial infectious diseases are caused by bacteria, fungi, spirochetes, viruses, etc., causing tissue destruction, nodules or tumor rupture. An vascular inflammatory ulcer caused by an immune abnormality is formed by necrosis of tissue due to arterial or arteritis. Circulatory or neurological dysfunction is a tissue disorder caused by dystrophic disorders such as varicose veins, leprosy, ulcers, etc.

an examination

Related inspection

General radiography examination CT examination

Squamous cell carcinoma occurs in the lower lip, tongue, nose, vulva, mostly at the junction of skin and mucous membranes, the edge of the ulcer is high, red hard, ring-shaped, cauliflower-like appearance, peripheral inflammatory reaction is significant, and there are many regional lymphadenopathy. .

Differential diagnosis

According to clinical manifestations, it is easier to diagnose malignant tumors. The diagnosis of squamous cell carcinoma depends on histopathology, while histopathologically requires poorly differentiated mucoepidermoid carcinoma with minimal mucus cells, necrotic parotid metaplasia. Identification.

1. Traumatic ulcers: There are many clear wounds and local irritants around the ulcer.

2. Herpetic gingivitis: The ulcers cluster and can fuse with each other to form larger ulcers with irregular edges.

3. Cancerous ulcers: Most of the cancerous ulcers are hyperplastic ulcers. The surface is protuberance-like, and the surface is surrounded by hard infiltration.

4. Tuberculous ulcer: The patient has a history of tuberculosis, the ulcer base has granular hyperplasia, the oozing exudate, and the edge can form an undercut.

5. Necrotic parotid metaplasia: necrotizing parotid metaplasia is a non-neoplastic inflammation that occurs in the small parotid gland, with ulceration on the edge of the ulcer, tenderness, and self-healing from weeks to months. Its pathological features are necrotic lobular necrosis, and acinar and glandular ducts are replaced by squamous cells.

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