Reticular dementia

Introduction

Introduction The intelligent damage of multiple infarct dementia sometimes involves only certain specific and limited cognitive functions, such as calculation and naming. The general reasoning and judgment can remain intact for a considerable period of time, and often can detect these obstacles and actively seek medical treatment or make efforts to make up for it. Therefore, it is called "net-like dementia".

Cause

Cause

Reasons for mesh-like dementia:

Because cerebral arteriosclerosis affects the blood supply to the brain, especially the dementia syndrome caused by recurrent cerebrovascular accidents. The microemboli or ischemic plaque of the external cerebral artery (carotid artery or cone base artery) causes the majority of small infarcts in the white matter center of the brain.

Examine

an examination

Related inspection

Brain Doppler Ultrasound (TCD) EEG

Diagnosis of reticular dementia:

General reasoning and judgment can remain intact for a considerable period of time, and often can detect these obstacles and actively seek medical treatment or make efforts to make up for it.

EEG often showed obvious abnormalities; cerebrospinal fluid examination may have a slight increase in protein; cerebral blood flow with the examination of reduced blood vessel elasticity, increased resistance, decreased blood flow and slow; CT scan showed low density area and localized ventricular enlargement; magnetic Resonance imaging can show a cavity infarction.

Diagnosis

Differential diagnosis

Differential diagnosis of mesh-like dementia:

Progressive dementia: a syndrome caused by a progressive, progressive brain disease. The course of the disease is progressive. Features are a variety of advanced cortical dysfunction, involving memory, thinking, orientation, understanding, calculation, judgment, speech and learning ability. Clear consciousness, poor self-control ability, social or motivational decline, often accompanied by cognitive impairment, but sometimes earlier than cognitive impairment.

Subcortical dementia: In general, multiple cerebral infarction dementia, thalamic dementia, and Binswanger disease are mostly in the subcortical nucleus and white matter, and the symptoms are mostly in the subcortical dementia range. Large-area cerebral infarction dementia and infarct dementia in the watershed area involve both cortex and subcortical, and the clinical symptoms and signs are cortical and subcortical mixed dementia.

Vascular dementia: Dementia caused by cerebral infarction due to vascular disease, including hypertensive cerebrovascular disease. Dementia can occur after multiple transient ischemic attacks or continuous acute cerebrovascular accidents, and individuals can also occur after a severe stroke. Infarcts are generally small, but the effects can be additive. It usually starts in old age, including multiple cerebral infarction dementia.

General reasoning and judgment can remain intact for a considerable period of time, and often can detect these obstacles and actively seek medical treatment or make efforts to make up for it.

EEG often showed obvious abnormalities; cerebrospinal fluid examination may have a slight increase in protein; cerebral blood flow with the examination of reduced blood vessel elasticity, increased resistance, decreased blood flow and slow; CT scan showed low density area and localized ventricular enlargement; magnetic Resonance imaging can show a cavity infarction.

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