Fibrous dysplasia

Introduction Poor fiber structure is a benign tumor-like differentiation of fibrous bone tissue. Commonly a single lesion of bone lesions. It often occurs in the rapid growth of adolescent bones and can be expanded throughout life. Multiple fibrous dysplasia is a multiple lesion adjacent to the bone. The most common sites are the proximal femur, the tibia, the tibia, the ribs and the head bones. Other bones also occur in the body. The lesion affects the strength of the bone, and the pathological fracture repeatedly causes malformation. Accompanied by sexual precocity, the multifocal fibrous dysplasia of the Café-au-lait plaque is called Albright's syndrome.

Cause

The cause of bone fiber dysplasia is unknown. Some people think that it is caused by internal spasm disorder, and some people think that it is formed by abnormal bone development.

an examination

Related inspection

Blood routine X-ray oil angiography

According to the clinical manifestations of age, location and other clinical features, especially in combination with the characteristics of the skull X-ray, diagnosis can be made. Check the trace elements of the body to see if there is a lack of iron, calcium and zinc, how to eat and sleep, whether there is abnormal pain, diarrhea, the inherent characteristics and essential properties of the fiber, determine the properties of the fiber, from micro to molecular composition, macro to fiber Morphology; There are many structural diversity and structural levels, clinical manifestations such as age and location of onset, combined with X-ray findings of the skull.

Differential diagnosis

1. Cyst type: more common in the early stage of the disease or lesions in the calvarial area, cystic bone density reduction areas of varying sizes can be seen between the skull barriers, and some are multi-atrial, widened, and outer plates The ridges are thinner and the inner panels are often unaffected.

2. Sclerosing type: more common in the late stage of the disease or in the bottom of the skull, the lesions are more extensive, often causing changes in the skull deformity, thickening of the bone, increased shadow density is "ivory" hardening changes, more common in the frontal bone The winglet of the plate and sphenoid bone.

3. Hybrid: Both cystic and sclerotic types exist at the same time, more common in the skull, and the smaller one needs to be differentiated from the skull changes caused by meningiomas. If it is multiple, the above-mentioned similar performance can be seen in the bones of other parts of the body. When there is cartilage tissue, it is cloud-like or cotton-like shadow. When there is more bone tissue, it can be ground glass.

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