Adrenal poisoning

Introduction Adrenalin and noradrenalin are commonly used first-aid drugs, and excessive amounts can cause poisoning. A small number of patients are highly sensitive to adrenaline, although adverse effects can also occur with therapeutic doses. Children with pre-excessive hyperthyroidism and cardiovascular disease are also sensitive to adrenaline and are prone to poisoning. The adrenal cortex can secrete a variety of important fat-soluble hormones. More than 20 kinds of fat-soluble hormones are known. They can promote non-sugar compounds (such as protein) or glucose metabolism in the body, maintain the balance between sodium and potassium ions in the body, and kidneys, muscles, etc. Features have an impact. Generally, it is not removed when the animals are slaughtered or the medulla is softened. It is lost when it is removed. It is eaten by people and the concentration of adrenaline in the body is increased, causing poisoning.

Cause

Adrenaline poisoning is caused by excessive dose of the drug; in addition, those who are allergic to this product may have serious adverse reactions in small doses.

Norepinephrine poisoning is caused by excessive use or excessive intravenous drip.

an examination

Related inspection

Urine routine renal angiography serum steroid binding -globulin

The side effects and toxic effects of norepinephrine are similar to adrenaline. When misused, it can cause extensive vasospasm. The child is pale, bruising at the ends of the limbs, headache, nausea, vomiting, abdominal pain, sweating, and ventricular Or supraventricular premature beats, sinus bradycardia, ventricular tachycardia, ventricular fibrillation, etc. The persistent excess can affect the blood circulation of the central nervous system and major organs such as heart and kidney, and the ischemic symptoms and functional damage of various systems appear, which aggravates the degree of shock. If the drug solution is leaked at the intravenous injection and the high concentration of the drug solution is applied, the extremity and the local necrosis of the injection may occur, and the child with shock is more likely to suffer from local drug stagnation.

Differential diagnosis

Mainly differentiated from other anti-shock drug poisoning. Dopamine poisoning, mainly nausea, vomiting, tachycardia, arrhythmia, dyspnea, headache, etc.; interstitial hydroxylamine poisoning, mainly showing fear, headache, irritability, skin flushing, sweating, trembling, nausea, vomiting, weakness of limbs Pale, pale chest pain, palpitations, small movements or slowness, decreased urine output, metabolic acidosis, high blood sugar, etc.

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