Spinal destruction

Introduction Spinal damage is divided into benign and malignant destruction. Reactive hyperplasia at the margin of destruction is mostly benign, mainly found in spinal tuberculosis and primary benign tumors. Destruction of edge blur without sclerosis is mostly malignant bone destruction, more common in metastatic tumors or primary malignant tumors. Spinal tuberculosis is often secondary to tuberculosis. Some patients may have no symptoms of tuberculosis. After pulmonary infection, blood can be transmitted to the whole body, and the spinal cord can cause spinal infection. The spinal blood supply is mostly terminal, and there is no blood circulation between the vertebral bodies. Cartilage disc, so the spinal tuberculosis is more common in the center type and the edge type.

Cause

Spinal tuberculosis is often secondary to tuberculosis. Some patients may have no symptoms of tuberculosis. After pulmonary infection, blood can be transmitted to the whole body, and the spinal cord can cause spinal infection. The spinal blood supply is mostly terminal, and there is no blood circulation between the vertebral bodies. Cartilage disc, so the spinal tuberculosis is more common in the center type and the edge type. In fact, tuberculosis can be obtained from bones in any part of the body, and tuberculosis in the spine is about 50%. Other joints such as knee joints and hip joints can also get tuberculosis.

an examination

Related inspection

Spinal MRI examination of the spine vertebral body

In terms of the way of vertebral destruction, it has been classified into edge type, central type, subperiosteal type and accessory type according to the X-ray plain film performance, but the flat piece is difficult to overcome the mediastinum, gastrointestinal gas and the like, and the CT can be clearly displayed. The way bone defects in the spine tuberculosis provide valuable information for developing treatment options.

Differential diagnosis

1, spinal bone hyperplasia: vertebral body or attachment local or all bone hyperplasia, increased bone density. Found in chronic lesions such as spinal degeneration, skeletal fluorosis, osteopetrosis, malformation osteitis, idiopathic hypertrophic osteoarthrosis, ankylosing spondylitis, and spinal tuberculosis healing.

2, vertebral morphological changes: including lateral or posterior deformity, skeletal rigidity, spinal canal enlargement, intervertebral foramen enlargement, increased vertebral arch spacing or abnormal reduction, vertebral wedge deformation, spinal dislocation and other changes.

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