Symptom >> Internal Medicine >> Gastroenterology

Liver shrinkage

Introduction
Cause
Examine
Diagnosis

Introduction

Introduction "Liver atrophy" refers to B-ultrasound or CT detection, liver volume shrinkage, belongs to the "bulging" of traditional medicine, modern medicine is characterized as "cirrhosis", liver reduction has no other effective treatment methods at home and abroad except liver transplantation. In the 1940s, the medical community first proposed the concept of fulminant hepatic failure (FHF), originally called acute liver atrophy or acute liver necrosis.

Cause

Cause

"Liver shrinkage" is also present in the late stages of hepatitis, but it is completely different from the etiology and pathogenesis of hepatomegaly:

First, the liver and kidney yin deficiency, the water is not good, the fire on the virtual fire, the liver is shrunk and shrunk.

The second is the lack of liver yin and the steaming of dampness and heat. The body fluid is swollen, and the liver is lost in nourishing, so it gradually shrinks.

Third, yin deficiency or yin deficiency and dampness lead to liver atrophy.

Examine

an examination

Related inspection

Liver ultrasound examination of liver volume

Different causes of liver shrinkage have different symptoms:

1, sputum, nose, blood, red lips, upset, insomnia, red tongue, little moss or no moss, dry stool, pulse string count.

2, dull complexion or sclera skin yellow staining, mouth bitter or dry mouth, upset and irritability, dry yellow urine or soft palate, thin tongue, yellow tongue or yellow greasy tongue.

3, dry lips, upset, insomnia, dreams, abdominal distension, ascites, thick tongue, white greasy fur, thin or soft stools, slow pulse.

Diagnosis

Differential diagnosis

Identification:

Hepatic sinus expansion: The liver volume increases and the surface is smooth in the acute phase, showing lymphatic vessel expansion and "liver crying" signs. Under the light microscope, the central vein and the inferior venous intima were significantly swollen, the lumen was narrowed or occluded, the blood flow was blocked, and the hepatic sinus was obviously dilated and congested with varying degrees of hepatocyte turbidity, swelling and necrosis. Hepatocytes disappeared in the area of necrosis, residual reticular fiber scaffolds, red blood cells infiltrated into the hepatic sinus and Dissel's space, and typical hemorrhagic necrosis changes.

In the subacute stage, the surface of the liver is reticulated, and the central vein and the inferior venous endothelium are hyperplasia and thickening, forming fibrosis and stenosis and occlusion. In this period, there may still be hepatic sinus expansion, hemorrhage and hemorrhagic hepatocyte necrosis, fibrosis in the central vein, and no false leaflet formation.

Progressive liver shrinkage: fulminant hepatic failure is a syndrome in which hepatic cell necrosis and severe liver function damage are caused by a variety of causes, and there is no history of liver disease and hepatic encephalopathy within 8 weeks after the disease. The onset is urgent, the progress is fast, and the mortality rate is high. Early diagnosis and early treatment can reduce the mortality rate. Early symptoms include jaundice, persistent hypothermia, low fever, gastrointestinal symptoms, bleeding tendency, progressive liver shrinkage, liver odor, flapping tremor, heart rate, hypotension, etc., and later symptoms are hepatic encephalopathy. Cerebral edema is characterized by convulsions, slow breathing, irregular rhythm, coagulopathy and hemorrhagic bleeding with skin, gums, nasal mucosa, bulbar conjunctiva and gastric mucosa.