Ether poisoning

Introduction to ether poisoning Most of the ether poisoning (etherpoisoning) is caused by excessive use of anesthesia, excessive concentration or dripping too fast, and occasional accidental drinking. Some patients are allergic to ether, and pregnant women are poisoned by ether, which can cause fetal or neonatal poisoning. basic knowledge The proportion of illness: 0.06% Susceptible people: no special people Mode of infection: non-infectious Complications: hematuria

Cause of ether poisoning

Poisoning is often caused by excessive dosage or dripping too fast. In addition, aspiration and accidental drinking are common causes of poisoning.

Ether poisoning prevention

1. Patients with lung disease, heart disease, liver and kidney dysfunction and those with a history of allergies are banned from ether.

2. Strengthen safety production to prevent run, run, leak, drip and pollution of production workshops and environment.

3. Human body protection

(1) Skin contact: Remove contaminated clothing and rinse with plenty of running water.

(2) Eye contact: Lift the eyelids and rinse with running water or saline. Seek medical attention.

(3) Inhalation: Quickly leave the site to fresh air and keep the airway open.

(4) Respiratory protection: Wear a filter-type gas mask when the concentration in the air exceeds the standard.

Ether poisoning complications Complications

"Ether convulsions" may occur, manifested as clonic or tonic shock, angular arch reversal, closed jaws, allergic to ether, diffuse redness of the skin, palpitations and difficulty breathing, kidney damage may occur Hematuria, proteinuria, tubular urine, urinary fistula, acetone and urine.

Symptoms of ether poisoning Common symptoms Nausea and vomiting Blood pressure drop Abdominal pain Pupil abnormal mouth and pharyngeal burning Sensation hypothermia Diarrhea Weekly sweating Breathing Slow slow complexion Violet

Symptoms of acute poisoning include vomiting, bruising complexion, decreased body temperature, dilated pupils, shallow and irregular breathing, and even sudden stop of breathing; or weak pulse rate, decreased blood pressure and even circulatory failure. When children inhaled ether vapor concentration exceeds 15% (When the baby is lower), it can directly act on the heart conduction system and even cause cardiac arrest.

Patients with lungs, kidneys, etc. or diabetics often have delayed poisoning after inhaling ether. Generally, they have uneasiness, convulsions, and pulse rate within 12 to 24 hours after anesthesia; or have jaundice, cyanosis, coma, convulsions. May die within a few days.

In moderate to deep ether anesthesia, increased secretion of adrenocortical hormone and norepinephrine due to stimulating effects may lead to increased blood pressure and even intracranial hemorrhage. In deep anesthesia, sometimes tonic or clonic convulsions occur. Often with angulation and closed jaw, such convulsions are more likely to occur in acute toxemia or sepsis, alkalosis or decreased blood calcium and long after anesthesia.

Oral ether poisoning, and mouth, tongue, throat, throat and stomach burning, runny, nausea, vomiting, abdominal pain, diarrhea, stomach dilatation, pulse fast, facial flushing, sweating, difficulty breathing, hematuria, convulsions, coma Wait.

When the therapeutic amount of ether is applied, allergic symptoms such as diffuse redness, paresthesia, palpitations and difficulty in breathing may occur.

Examination of ether poisoning

Laboratory inspection

When ether is poisoned, the remaining poisons or specimens that may be toxic, such as vomit, stomach contents, urine, feces, blood specimens, etc., should be routinely reserved. Perform toxicological analysis or bacterial culture if necessary. Although the toxicant analysis is important, it cannot wait until the test results are reported before starting treatment. For chronic poisoning, checking for the presence of poisons in the environment and in the human body can help to determine the diagnosis.

Diagnosis and identification of ether poisoning

Primary ether poisoning should be differentiated from the following toxic diseases:

Alcoholism is commonly referred to as drunkenness. Alcohol (ethanol) drinking a large amount of alcoholic beverages at one time will have an excitatory and post-inhibitory effect on the central nervous system. Severe poisoning can cause respiratory and heartbeat inhibition and death. Symptoms: 1, nausea, vomiting, dizziness, slang, swaying. 2, severe coma, incontinence, respiratory depression.

Clinical manifestations of naphthalene poisoning:

(1) Acute poisoning:

1) Inhalation poisoning: 1 eye and respiratory mucosal irritation; 2 headache, fatigue, nausea, vomiting, optic neuritis, etc.; 3 low back pain, frequent urination, hematuria, proteinuria, etc.; 4 severely ill patients with jaundice, hemoglobinuria and liver damage, There are even convulsions and coma.

2) Oral poisoning: nausea, vomiting, abdominal pain, diarrhea, hepatomegaly; chills, fever, low back pain, soy sauce urine, hemolytic anemia and jaundice; severe acute renal failure, liver necrosis.

(2) Chronic effects.

(3) laboratory tests may be abnormal in critically ill patients: 1 hematuria, proteinuria, hemoglobinuria; 2 blood routine: low hemoglobin, increased reticulocyte; 3 liver and kidney damage.

The clinical manifestations of lead poisoning are:

1, neurasthenic syndrome symptoms appear earlier, more common, headache, dizziness, fatigue, limb pain.

2, dyspepsia patients with metallic taste, abdominal pain, constipation, a small number of patients in the gingival margin mucosa can be seen in the formation of "lead line" formed by the deposition of lead sulfide particles, dark gray or blue banded or irregular plaque Often seen in a few poor oral hygiene.

The material in this site is intended to be of general informational use and is not intended to constitute medical advice, probable diagnosis, or recommended treatments.