Poetry-like language

Introduction A poetic (or sub-section) speech: another feature of speech impairment in the damage of the cerebellum system. Due to the abnormal configuration of the accent when speaking, it is evenly divided into many inconsistent speech stages. It is very similar to the tone of the old-fashioned poetry. The poetry-like language is most commonly seen in the cerebellar palsy and cerebellar degenerative diseases. 10% to 15% of patients with multiple sclerosis have such dysarthria and have a poetry-like language.

Cause

The poetry-like language is most commonly seen in the cerebellar palsy and cerebellar degenerative diseases.

10% to 15% of patients with multiple sclerosis have such dysarthria and have a poetry-like language.

The cause of fertility language disorders is currently inconclusive. Some experts believe that it is related to genetic factors; some experts believe that it is the result of the combination of biological factors and environmental factors in the development of the brain. If the family's psychological environment, language environment and poor parenting style are in the early stage of child development, it may also lead to the development of children's language ability to a certain extent.

It should be noted that children with developmental language disorders are slow to respond to the language, and respond to other sounds normally and have inquiry behaviors. The understanding and imitation ability of music is normal; their inner language function is normal, and they can interact with the dolls. Play games; normal interpersonal skills, knowing to express your emotions and needs with expressions, gaze or actions, attachment to the mother, being able to get along with the children; IQ is also within the normal range.

an examination

Related inspection

Tone and voice language check

1. First symptom: including one or more limbs with local numbness, tingling or single limb instability, single eye sudden vision loss or blurred vision (opic neuritis), double vision, balance disorder, bladder dysfunction (urine urgency) Or poor urine flow, etc., some patients show acute or progressive progression of spastic paraplegia and loss of sensation. These symptoms usually last for a short period of time and disappear after a few days or weeks, but some residual signs can still be found after careful examination.

2. After the first onset, there may be several months or years of remission, and new symptoms or recurrence of the original symptoms may occur. Infection can cause recurrence, and women are more likely to relapse about 3 months after delivery. The increase in body temperature can temporarily worsen the stable condition. The number of relapses can be as many as 10 times or more. After multiple recurrences and incomplete remission, the patient's weakness, stiffness, sensory disturbance, limb instability, visual impairment and urinary incontinence can be heavier and heavier.

3. Clinical common symptoms and signs: MS patients with more signs than symptoms are important clinical features, the patient complained of one side of lower limb weakness, gait instability and numbness, but may find bilateral pyramidal tract signs or Babinski sign. The coexistence of nystagmus and internuclear ophthalmoplegia is indicated as a brainstem lesion and is a two-symptom of MS.

(1) Limbs are more common, and common asymmetry is mild and paraplegic, showing weakness or heavy feeling of lower limbs.

(2) About half of the cases can be seen with visual impairment, starting from one side and invading the other side at intervals, or two eyes are involved in a short time. The onset is more urgent, often with multiple remissions - recurrence, which can begin to recover after a few weeks.

(3) The nystagmus is mostly horizontal or horizontal plus rotation, and diplopia accounts for about 1/3. Invasion of the medial longitudinal bundle causes internuclear ophthalmoplegia, invasion of the anterior reticular formation (PPRF) leads to a semi-syndromic; other cranial nerve involvement is rare, such as central or peripheral facial paralysis, deafness, tinnitus, dizziness, biting Weak muscles, dysarthria, and difficulty swallowing.

(4) More than half of patients have sensory disturbances, including deep sensory disturbances and Romberg signs.

(5) Ataxia is seen in about half of the cases, but Charcot's three main signs (eye, intentional tremor, and sinusoidal language) are only found in some patients with advanced MS.

(6) Neurophysiological examination confirmed that MS can be combined with peripheral nerve damage (such as polyneuropathy, multiple mononeuropathy), may be due to peripheral nerve P1 protein and the central nervous system MBP as the same component, both demyelination To.

(7) There may be pathological emotions such as euphoria and excitement. Most cases show depression and irritability, as well as mental disorders such as apathy, lethargy, strong crying, slow response, repeated language, suspicion and persecution.

Optic atrophy, nystagmus, and dysarthria are often found in advanced case examinations, and pyramidal tract signs, sensations, or cerebellar signs may occur in some or all of the limbs. It has been confirmed that certain symptoms are extremely rare in MS, such as aphasia, hemianopia, extrapyramidal dyskinesia, severe muscle atrophy, and fasciculation, which are often used as exclusion criteria for MS.

Differential diagnosis

Symptoms of esoteric language confusion:

Another manifestation of the difference in articulation difficulties with cerebellar system damage:

Outbreak speech: Speech is significantly prolonged. There is uneven sound intensity. Therefore, it is often violent. The patient's speech intensity is sometimes extremely low. Sometimes it is extremely high. And quickly issued a series of syllables or words.

The configuration of the accent when speaking is abnormal and is evenly divided into many inconsistent speech stages. It is very similar to the tone of the old-fashioned poetry. The poetry, intentional tremor and nystagmus together constitute the classic Charcot triad.

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