High postprandial blood sugar

Introduction The second clinical manifestation of type 2 diabetes is that the resistance is aggravated. Although hyperinsulinemia, but the receptor is less sensitive, there is still postprandial hyperglycemia; it is characterized by normal fasting blood glucose but elevated postprandial blood glucose.

Cause

Insulin resistance is prevalent in type 2 diabetes, accounting for 90%, and is a major cause of type 2 diabetes.

Factors associated with type 2 diabetes:

Genetic factors: Similar to type 1 diabetes, type 2 diabetes is also characterized by family onset. It is therefore likely to be related to genetic inheritance. This genetic property is more pronounced in type 2 diabetes than in type 1 diabetes.

For example, one of the twins has type 1 diabetes and the other has a 40% chance of developing the disease; but in the case of type 2 diabetes, the other has a 70% chance of developing type 2 diabetes.

Obesity: An important factor in type 2 diabetes may be obesity. Genetic causes can cause obesity and can also cause type 2 diabetes. Excess fat in body-centered obese patients is concentrated in the abdomen, and they are more likely to develop type 2 diabetes than those whose fat is concentrated on the buttocks and thighs.

Age: Age is also a contributing factor to type 2 diabetes. Half of patients with type 2 diabetes often develop after 55 years of age. The prevalence of diabetes in older patients is also associated with the overweight of older people.

Modern lifestyle: Eating high-calorie foods and reducing exercise can also cause diabetes, which is thought to be caused by obesity. Obesity, like type 2 diabetes, is more prevalent among Asian American and Latin American businessmen whose diet and activity habits have been westernized.

an examination

Related inspection

Blood sugar and blood lipid test

Insulin resistance is prevalent in type 2 diabetes, accounting for 90%, and is a major cause of type 2 diabetes. Most scholars believe that the insulin resistance system is the primary, but it is likely that both "insulin resistance" and "secretory disorder" exist, but the performance is sequential, and the severity is different.

Divided into three phases:

In the first phase, there was resistance and hyperinsulinemia, and plasma glucose was normal; its blood sugar was normal but blood lipids were abnormal.

In the second phase, although the resistance is aggravated, although hyperinsulinemia is present, the more insensitive the receptor, the postprandial hyperglycemia still occurs; it is characterized by normal fasting blood glucose but elevated postprandial blood glucose.

In the third phase, resistance still exists, but insulin secretion is reduced, leading to fasting hyperglycemia. Postprandial and fasting blood glucose increased above normal.

Differential diagnosis

The sugar in the blood is called blood sugar, and in most cases it is glucose. Most of the energy required for cell activity in various tissues in the body comes from glucose, so blood sugar must be maintained at a certain level to maintain the needs of various organs and tissues in the body. Normal people have a fasting blood glucose concentration of 80-120 mg/dl in the morning. Fasting blood glucose concentrations above 130 mg/dl are called hyperglycemia. If the blood glucose concentration exceeds 160-180 mg/dl, a part of glucose is excreted in the urine, which is diabetes. Elevated blood sugar is common in diabetes. Hyperglycemia refers to a fasting blood glucose above the upper limit of 7.3 mmol/L (130 mg/dl).

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