Stiff heart

Introduction This is Dr. Benjamin Levine of the Dallas Institute of Sports and Environmental Medicine and colleagues who examined 12 sedentary seniors, 12 excellent athletes in their 60s, and a group of sedentary older than half of the elderly. adult. By scanning the racing records, they found excellent athletes engaged in track and field, land-based and cycling, and selected subjects over the age of 65 who consistently triumphed in the race. Researchers bet that these older athletes are comfortable and well-preserved for decades of sustained and endurance exercise, so they may prevent the so-called "hardening" of the heart caused by old age and maintain left ventricular compliance, possibly avoiding diastolic heart failure. .

Cause

Failure to get good exercise or long-term non-exercise is an important cause of heart stiffness.

1. Cholesterol is too high, and the chance of suffering from heart disease is three times higher than that of ordinary people, because too much cholesterol in the body will accumulate in the blood vessels, making the blood vessels narrower and obstructing blood circulation.

2. Smokers are two and a half times more likely than ordinary people. The reason is that nicotine or tobacco chemicals in cigarettes can damage the blood vessels of the heart. If blood vessels crack, cholesterol will accumulate.

3, high blood pressure, two and a half times more than the opportunity of ordinary people, high blood pressure will cause vasoconstriction.

4, diabetes, female patients have twice the chance of having heart disease, and 50% more men.

5, excessive obesity, because obesity leads to high blood pressure, high blood fat, diabetes, and these diseases can induce heart disease.

6, life is tight, nervousness makes the arrhythmia, endocrine disorders, affecting the heartbeat, stimulating heart attack, the normal heart is bigger than the fist, pumping blood through the circulatory system. The heart beats an average of 100,000 times a day and pumps about 2,000 gallons of blood back and forth. If you calculate the life of 70 years old, your heart rate can reach 2.5 billion times.

an examination

Related inspection

ECG dynamic electrocardiogram (Holter monitoring)

The clinical symptoms can occur in patients with normal left ventricular ejection fraction (LVEF). This view has been widely accepted. It is now called the heart failure wieh normal left ventricular ejection fraction (HFNEF). . In some cases, the patient's clinical performance is as severe as in patients with low ejection fraction heart failure, such as patients admitted to hospital for acute pulmonary edema. Regarding the clinical features of patients with HFNEF, stable HFNEF patients have approximately the same changes in physiological and neurohormonal levels as those with low ejection fraction, including decreased peak oxygen consumption and elevated circulating neurohormonal levels such as (brain natriuretic peptide (BNP) and Norepinephrine).

Differential diagnosis

Cardiac hypertrophy: There are many reasons, including hypertensive left ventricular hypertrophy, septic heart failure, anterior cardiac infarction, athlete's heart, biceps countercurrent, aortic stenosis, proliferative obstructive cardiomyopathy, pulmonary hypertension, cardiopulmonary Disease, dilated cardiomyopathy, endocarditis, pericardial hydrocephalus, left ventricular aneurysm, biceposis, etc. When doctors do physical examinations, they sometimes notice the phenomenon of heart hypertrophy. Into an electrocardiogram and chest X-ray photography, most of the heart hypertrophy can be diagnosed. For a more accurate diagnosis, cardiac ultrasound is the most accurate.

Pericardial tamponade: The pericardial cavity is the space between the parietal pericardium and the visceral pericardium on the surface of the heart. A small amount of light yellow liquid in the normal pericardial cavity lubricates the surface of the heart. Traumatic heart rupture or pericardial vascular injury caused by blood accumulation in the pericardial cavity called blood pericardial or pericardial tamponade, which is the cause of rapid death of cardiac trauma. M-mode ultrasound showed the ventricle activity curve of the pericardium. When the pericardial tamponade, the direction of the anterior wall of the right ventricle changed, and the diastolic phase showed a centripetal movement, that is, the posterior displacement.

Cardiac deposit amyloid: A disease in which myocardial dysfunction caused by amyloid protein deposition in myocardial tissue. Almost all patients have abnormal electrocardiogram, including low voltage, ST-T changes, sinus tachycardia, arrhythmia with atrioventricular block, and bundle branch block, premature beat, atrial fibrillation, Occasionally Q wave or myocardial infarction pattern.

Cardiac malformation: more common in congenital heart malformations, congenital heart malformations are congenital heart disease, one of the common malformations of the fetus, the incidence rate is about 0.4-0.8%. Most chromosomal abnormalities of the fetus, combined with many fetuses with severe deformities, have a cardiac malformation. At present, congenital heart disease accounts for the first cause of death in newborns and children. The easiest and safest method for prenatal diagnosis of congenital heart malformation is fetal echocardiography. Because prenatal observation of the fetal heart is affected by many factors, and prenatal echocardiography has certain limitations, the prenatal diagnosis rate of congenital heart malformation is currently only maintained at about 40%. Increasing the prenatal diagnosis rate of congenital heart malformation is one of the methods to reduce neonatal mortality and improve the survival rate of perinatal children.

The clinical symptoms can occur in patients with normal left ventricular ejection fraction (LVEF). This view has been widely accepted. It is now called the heart failure wieh normal left ventricular ejection fraction (HFNEF). . In some cases, the patient's clinical performance is as severe as in patients with low ejection fraction heart failure, such as patients admitted to hospital for acute pulmonary edema. Regarding the clinical features of patients with HFNEF, stable HFNEF patients have approximately the same changes in physiological and neurohormonal levels as those with low ejection fraction, including decreased peak oxygen consumption and elevated circulating neurohormonal levels such as (brain natriuretic peptide (BNP) and Norepinephrine).

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