Tachycardia

Introduction Adults have a heart rate of more than 100 beats per minute. The tachycardia is divided into two types: physiological pathology. When running, drinking, heavy physical labor and emotional agitation, the heart rate is accelerated to physiological tachycardia; if high fever, anemia, hyperthyroidism, hemorrhage, pain, hypoxia, heart failure and cardiomyopathy cause tachycardia, called pathological heartbeat Overspeed. Physiological tachycardia may not be too tight, but pathological tachycardia should be treated in hospitals.

Cause

Physiological factors: such as agitation, exercise, etc.

Disease factors: such as abnormal cardiac conduction, viral myocarditis, hyperthyroidism and other factors such as lack of exercise, mental and other factors, currently only need symptomatic support treatment, with diet and moderate exercise to adjust.

Pathological tachycardia can be divided into sinus tachycardia and paroxysmal supraventricular tachycardia. The heart position of sinus tachycardia is characterized by a gradual increase in heart rate and slowing down. Usually, the heart rate does not exceed 140 times per minute. Most of the patients have no organic disease, usually no obvious discomfort, sometimes flustered, shortness of breath, etc. symptom. Paroxysmal supraventricular tachycardia can reach 160 to 200 beats per minute. It is characterized by sudden onset and sudden stop, which can occur in patients with organic lesions or cardiac organic lesions. Suddenly, the patient suddenly felt palpitation and increased heart rate. It lasted for several minutes, hours to days, and suddenly returned to normal heart rate. At the time of the attack, the patient felt guilty, chest tightness, discomfort in the precordial area, and swelling of the head and neck, and a sense of jumping. People without heart disease generally have no significant impact, but the attack time is long. When the heart rate is more than 200 beats per minute, the patient has black eyes, dizziness, fatigue, nausea and vomiting, and even sudden fainting and shock. Patients with coronary heart disease have tachycardia and often induce angina.

an examination

Related inspection

Dynamic electrocardiogram (Holter monitoring) Electrocardiogram Ultrasound examination of thyroid and parathyroid glands 4-hydroxy-3-methoxy mandelic acid urinary catecholamine

You can go to the hospital to have an electrocardiogram and exclude the atrioventricular bypass (pre-excitation syndrome). Pathological tachycardia can be divided into sinus tachycardia and paroxysmal supraventricular tachycardia. Sinus tachycardia is characterized by a gradual increase in heart rate and slowing down. Usually, the heart rate does not exceed 140 times per minute. Most patients without cardiac organic disease usually have no obvious discomfort, and sometimes have symptoms such as palpitation and shortness of breath. Paroxysmal supraventricular tachycardia can reach 160 to 200 beats per minute. It is characterized by sudden onset and sudden stop, which can occur in patients with organic lesions or cardiac organic lesions. Suddenly, the patient suddenly felt palpitation and increased heart rate. It lasted for several minutes, hours to days, and suddenly returned to normal heart rate. At the time of the attack, the patient felt guilty, chest tightness, discomfort in the precordial area, and swelling of the head and neck, and a sense of jumping. People without heart disease generally have no significant impact, but the attack time is long. When the heart rate is more than 200 beats per minute, the patient has black eyes, dizziness, fatigue, nausea and vomiting, and even sudden fainting and shock. Patients with coronary heart disease have tachycardia and often induce angina.

Differential diagnosis

Healthy people's exercise and emotional stress can cause tachycardia.

General tachycardia is supraventricular, such as sinus tachycardia. The ventricular is a life-threatening heart rhythm change, which will be lost if it is not treated immediately more than 160 times/min. Therefore, we must first understand the classification of arrhythmia (the ECG can be diagnosed) before it can be processed.

It is not surprising that adolescents have arrhythmia, mostly physiological. Occasionally, the heart beats so badly that it is estimated that the heart rate is increasing, and it is generally not necessary. You can go to the hospital to have an electrocardiogram and exclude the atrioventricular bypass (pre-excitation syndrome). Pathological tachycardia can be divided into sinus tachycardia and paroxysmal supraventricular tachycardia. Sinus tachycardia is characterized by a gradual increase in heart rate and slowing down. Usually, the heart rate does not exceed 140 times per minute. Most patients without cardiac organic disease usually have no obvious discomfort, and sometimes have symptoms such as palpitation and shortness of breath. Paroxysmal supraventricular tachycardia can reach 160 to 200 beats per minute. It is characterized by sudden onset and sudden stop, which can occur in patients with organic lesions or cardiac organic lesions. Suddenly, the patient suddenly felt palpitation and increased heart rate. It lasted for several minutes, hours to days, and suddenly returned to normal heart rate. At the time of the attack, the patient felt guilty, chest tightness, discomfort in the precordial area, and swelling of the head and neck, and a sense of jumping. People without heart disease generally have no significant impact, but the attack time is long. When the heart rate is more than 200 beats per minute, the patient has black eyes, dizziness, fatigue, nausea and vomiting, and even sudden fainting and shock. Patients with coronary heart disease have tachycardia and often induce angina.

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