Sore hands

Introduction Hand acid is divided into two types, one is temporary hand acid, the other is persistent hand acid, which refers to the acidulence discomfort of the hand. Calcium in plasma and body fluids is mainly present in combination with calcium and free calcium. The former is mainly combined with albumin, and a small amount is combined with organic acids, such as calcium citrate, calcium lactate, calcium phosphate and the like. Free exchange of calcium with bound calcium is in a dynamic equilibrium, which is mainly affected by pH. In the case of acidemia, free calcium (Ca2+) increases and vice versa. In addition, a certain product is maintained between blood calcium and blood phosphorus concentration, that is, [Ca] × [P] = 350 to 400 mg / L. Only free calcium really has the physiological function of calcium.

Cause

1, such as long-term typing, playing the piano and so on and causing hand acid, it is because the nerves of the hand joints are in a tight state at any time, without the temporary hand acid produced by the rest.

2, excessive hand movement or heavy hand caused by hand acid caused by temporary hand acid, such as playing badminton, the hand acid caused by table tennis, it is because there is no good exercise for a long time, all of a sudden too strong The reasons mainly include the following aspects: 1 Lactic acid accumulation. 2 muscle spasms. 3 muscle fiber or connective tissue damage. 4 other factors. If the osmotic pressure between tissues changes or acute inflammation, it can also cause muscle soreness.

3, hand acid may also be caused by calcium deficiency, no obvious cause of the pain of the arm, consider the sleep body pressure caused by the nerve inside the arm.

an examination

Related inspection

EEG examination electrocardiogram

Hand acid that is associated with calcium deficiency requires further examination to determine the cause of early treatment. Exercise acid acid can be improved in two or three days, and posture acidity can change the wrong posture. Diagnosis is not difficult with hypocalcemia caused by vitamin D deficiency, pancreatic disease or kidney failure. However, to distinguish between primary hypoparathyroidism and pseudohypoparathyroidism, blood PTH levels and PTH stimulation test imaging are needed to detect basal nodule calcification. ECG shows ST segment lengthening, QT interval prolongation and T wave abnormality, and even EEG abnormalities may occur.

Differential diagnosis

Differential diagnosis of hand acid:

1, hand cramps: hand cramps may be due to arteriosclerosis, high blood lipids, calcium deficiency or heart disease.

2, finger numbness: the sensory nerve of the finger is distributed by the cervical spinal cord to the hands and fingers, when some parts of the nerve damage, inflammation, tumor, pressure, etc. cause abnormalities, it will There is numbness in the fingers.

Hand acid that is associated with calcium deficiency requires further examination to determine the cause of early treatment. Exercise acid acid can be improved in two or three days, and posture acidity can change the wrong posture.

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