Hyperkeratosis of pores

Introduction The so-called pore keratosis, as the name suggests, is a problem caused by excessive keratosis of the pores; the patient has a small red rash with rough pores because the pores are blocked by the keratinized keratin. In general, the incidence of keratosis in middle-aged women is the highest; because it is a skin disease with genetic characteristics, depending on the family constitution. As for the location, it is most common under the arms, calves, thighs or under the cheeks. Pore keratosis is a constitution caused by innate genes. Some experts said that people with this constitution, the keratin around the hair follicle will thicken, causing the hair follicle mouth to be blocked by thick keratin, resulting in a small bulge.

Cause

At present, the true cause of the disease is not known, but most of the literature believes that the disease has a genetic predisposition (common in patients with ichthyosis), and occurs in people with allergic or atopic physique, or malnutrition (especially vitamins) A lack of people); if the environment is low in humidity, dry air, and certain irritants such as tar, oil, etc., may also lead to more common.

Pore keratosis is a constitution caused by innate genes. Some experts said that people with this constitution, the keratin around the hair follicle will thicken, causing the hair follicle mouth to be blocked by thick keratin, resulting in a small bulge. Some patients with atopic dermatitis and ichthyosis often have psoriasis. The symptoms in winter are more serious, because it is related to pore keratosis and thickening of keratin, so when the climate is dry and cold, the keratin thickening will be aggravated, which will make the symptoms more obvious. In addition to congenital constitutional factors, there are reports of vitamin A deficiency. Lack of vitamin A causes systemic dryness, and dryness exacerbates the symptoms of pore keratosis.

an examination

Related inspection

Skin test skin color

Clinically, it will be more obvious from the age of ten to the time of puberty, and the symptoms will gradually decrease with age. It is easier to get on the sides of the cheeks and the lateral areas of the limbs, such as the outside of the arm, the outside of the thigh or the buttocks. Skin color or brownish red papules can be seen in the affected area, most of which have no obvious symptoms, but sometimes accompanied by itching.

The pores are slightly reddened, and even when touched, they will feel a grain of one bulge. This is pore keratosis. To be more serious, the pores will be darker in color, dark red or brown, and the particles will be rougher and look like goose bumps. The most common part of it is on the outside of the upper arm, thighs, neck, and some people, even the entire back, cheeks, will have symptoms of pore keratosis.

In general, pore keratinization is not inconvenient except for its poor appearance. But most afraid of the patient to grab, because once the hand to go, it is the beginning of a terrible nightmare. When the patient scratches, the tissue around the pores will be edema. As a result, the pore openings become smaller and more likely to clog, resulting in acne and folliculitis. If scratched, scars and pigmentation will form. It is even more difficult and more troublesome to treat or improve at that time.

Differential diagnosis

Differential diagnosis of hyperporosis of the pores:

Mild cases should be differentiated from seborrheic dermatitis, and localized linear lesions should be identified with sputum.

1. The need to identify the following diseases, the identification points are as follows:

(1) Acanthosis nigricans: It is a soft papilloma-like papule, which is mostly confined to the folds of the neck, ankle, groin, etc., often combined with visceral cancer.

(2) Congenital reticular papillomatosis: The lesion is a flat, large papule, often confined to the upper part of the trunk.

(3) squamous keratosis: often a single sacral nodule of the head or neck.

2. In histopathology should be identified with the following diseases:

(1) Solar keratosis often has an interphase change in the epidermal cell nucleus.

(2) Chronic benign familial pemphigus: no fissures, but a layer of loosened bullae can be seen on the basal layer.

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