Irregular hardening of the articular surface

Introduction In the late stage of articular cartilage necrosis caused by various reasons, the joint surface is irregularly hardened and the gap is narrow and uneven. Common femoral heads, tibial tuberosity, vertebral sacs, etc., lesions occur in the joints with large mobility and weight. In the late stage of articular cartilage necrosis caused by various reasons, the joint surface is irregularly hardened and the gap is narrow and uneven. Pathological findings: This sign caused by smooth thickening and hardening of the bony joint surface is the result of absorption, mechanization and reactive bone hyperplasia by cartilage tissue after cartilage and osteonecrosis. Many systematic X-ray follow-up of chronic joint diseases have proved that this process is objective and regular. There is a conceptual understanding here that from the changes of X-ray signs, it is more and more obvious than the initial changes of joint disease; from the pathological section, necrotic cartilage is increasingly absorbed and repaired.

Cause

In the later stages of various chronic joint infections, articular cartilage degeneration or late osteochondritis, adult Kashin-Beck disease, soot pollution, fluorosis, and decompression sickness and hemophilia. Trauma, steroid therapy, alcoholics, idiopathic femoral head necrosis, radiation therapy, systemic lupus erythematosus. Trauma theory ----- the defects of the bone structure itself and a variety of factors, trauma is a direct cause. Pathological process of necrosis, repair, regeneration and recovery.

an examination

Related inspection

Joint examination blood test

X-ray examination of the joints helps to diagnose the disease.

X-ray performance: It is completely different from the signs of smooth and thickening of the bony joint surface. In the late stage of articular cartilage necrosis caused by various reasons, the joint surface is irregularly hardened and the gap is narrow and uneven. Pathological findings: This sign caused by smooth thickening and hardening of the bony joint surface is the result of absorption, mechanization and reactive bone hyperplasia by cartilage tissue after cartilage and osteonecrosis. Many systematic X-ray follow-up of chronic joint diseases have proved that this process is objective and regular. There is a conceptual understanding here that from the changes of X-ray signs, it is more and more obvious than the initial changes of joint disease; from the pathological section, necrotic cartilage is increasingly absorbed and repaired.

Differential diagnosis

Irregular sclerosis of the articular surface needs to be identified as follows.

First, rheumatoid arthritis: also known as rheumatoid arthritis (RA), is a chronic systemic inflammatory disease whose etiology is not yet known. Rheumatoid arthritis is currently recognized as an autoimmune disease.

2. Psoriatic arthritis: is an inflammatory joint disease associated with psoriasis. It has a psoriatic rash and causes pain, swelling, tenderness, stiffness and movement disorders in the joints and surrounding soft tissues. Some patients may have convulsions. Arthritis and/or spondylitis, prolonged course, easy recurrence, and advanced joint stiffness, leading to disability.

Third, pseudo gout: for the calcium pyrophosphate crystals deposited in the articular cartilage, synovium, capsule, ligament and cause local joints (which are more common in the knee) swelling, X-ray indicates that the articular cartilage surface has calcification lines, joints Crystals of calcium pyrophosphate can be found in the solution.

Fourth, osteoarthritis: also known as degenerative joint disease, proliferative osteoarthritis, different names derived from the pathological manifestations of joint disease that there is cartilage degeneration accompanied by the formation of new bone. The incidence of this disease increases with age, and it is a joint disease of the elderly who grow older.

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