Indigestion in newborns

Introduction to neonatal dyspepsia Neonatal dyspepsia refers to the symptoms of digestive dysfunction such as upper abdominal pain, bloating, early satiety, belching, anorexia, heartburn, acid reflux, nausea, vomiting, etc., which persists or recurs. Sexual diseases are the most common clinical syndrome in children's gastroenterology. basic knowledge Sickness ratio: 0.1%-0.5% Susceptible people: young children Mode of infection: non-infectious Complications: pediatric malnutrition pediatric malnutrition

Causes of neonatal dyspepsia

It is currently considered to be the result of a combination of multiple factors. Including diet and environment, gastric acid secretion, dysfunction of the digestive tract, psychological factors and some other gastrointestinal disorders.

Neonatal dyspepsia prevention

1. Try to avoid taking medicine that is irritating to the stomach. Aspirin, ibuprofen, etc.

2, adjust the type of food, reasonable diet structure, establish a good living habits. Do not picky eaters, diet should be moderate, eat less cold food.

3. Keep your child's mood comfortable during meals and avoid psychological stress.

Neonatal dyspepsia complications Complications, pediatric malnutrition, dystrophy, weight loss

Long-term dyspepsia can cause diseases such as malnutrition and anemia in children.

Neonatal dyspepsia symptoms Common symptoms Anorexia pediatric eating vomiting diarrhea, bloating, vomiting, gastroesophageal reflux symptoms

Clinical symptoms mainly include upper abdominal pain, abdominal distension, early satiety, hernia, anorexia, heartburn, nausea and vomiting. Symptoms can be recurrent and can be asymptomatic for quite some time. Or a symptom based on the onset of the disease, there may be a superposition of multiple symptoms. The cause of causing or aggravating the condition is unclear. Functional dyspepsia is divided into 4 types: dyskinesia type; reflux type; ulcer type; non-specific type.

1, dyskinesia dyspepsia

This type is mainly bloating, early satiety, and hernia. Symptoms are often aggravated after eating. Abdominal pain, nausea and even vomiting can occur when you are full. Gastric kinetics in 50% to 60% of children with proximal and distal gastric systolic and diastolic disorders.

2, reflux dyspepsia

The outstanding performance is post-sternal pain, heartburn, and reflux. Endoscopy showed no esophagitis. However, 24h hour pH monitoring revealed partial gastroesophageal reflux in some children. For patients with acid-free reflux, such symptoms are thought to be associated with increased acid sensitivity to the esophagus.

3, ulcerative dyspepsia

Its performance is the same as that of duodenal ulcer. It can be relieved by nighttime pain, hunger pain, eating or taking antacids. It can be accompanied by acid reflux. A few children have heartburn and the symptoms are chronic. No endoscopic examination revealed ulcers and erosive inflammation.

4, non-specific dyspepsia

Clinical manifestations can not be classified into the above types, often combined with irritable bowel syndrome.

In addition to reflux dyspepsia, several other classifications have no important clinical significance. Many children are not only classified into one subtype, but have little guidance for the treatment of children.

Neonatal indigestion check

1, laboratory inspection

Blood routine, liver, kidney function, blood glucose, thyroid function test, fecal occult blood test and gastroesophage 24-hour pH monitoring.

2, auxiliary inspection

Upper gastrointestinal endoscopy; liver, gallbladder, pancreatic B-mode ultrasound; chest X examination. Barium meal examination; radionuclide gastric emptying examination, gastrointestinal pressure measurement, etc.

Diagnosis and diagnosis of neonatal dyspepsia

diagnosis

1, clinical manifestations: chronic upper abdominal pain, abdominal distension, early satiety, hernia, acid reflux, heartburn, nausea, vomiting feeding difficulties and other upper gastrointestinal symptoms, lasting at least 4 weeks.

2, auxiliary examination: endoscopic examination found no gastric, duodenal ulcer, erosion, tumor and other organic lesions, no esophagitis. B-ultrasound and X-ray examination to exclude liver, gallbladder and pancreatic diseases.

3, laboratory tests for liver function, bilirubin, blood sugar and so on.

4. No diabetes, connective tissue disease, kidney disease and mental illness.

5, no history of abdominal surgery.

Identification

1, gastroesophageal reflux

The reflux subtypes of gastroesophageal reflux and functional dyspepsia are difficult to identify, and the clinical manifestations of gastroesophageal reflux are severe. Vomiting, hematemesis and blood in the stool, and swallowing pain after eating. Endoscopy confirmed varying degrees of esophageal inflammatory changes, and 24-hour esophageal pH monitoring showed an acid response.

2, upper gastrointestinal ulcer

Gastrointestinal ulcers include: stomach, duodenal ulcer, pyloric canal ulcer, anterior pyloric ulcer, erosive antral sinusitis. Endoscopic examination revealed extensive mucosal congestion, edema, erosion, and bleeding.

3, stomach cramps

Clinical symptoms are similar to functional dyspepsia, which is followed by systemic, digestive tract diseases or disorders that induce gastric emptying after surgery. The most common causes of gastroparesis are diabetes, uremia, and connective tissue disease. The diagnosis is determined by radionuclide gastric emptying function assay.

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