Cyanotic-digital clubbing-hepatopathy syndrome
Introduction
Aster - clubbing - liver disease syndrome Purpura-sickle-hepatic syndrome refers to a clinical manifestation of purple and blue skin and facial masks due to increased hemoglobin in the blood or abnormal blood hemoglobin derivatives such as methemoglobin and sulphide hemoglobin. Bun can appear on the whole body skin and my membrane, but at the end of the blood circulation, such as the lips, which are thinner in the skin, less pigmented, and rich in capillaries. Tongue, oral membrane, nose, cheeks, earlobe, nail bed, etc. are easier to observe. basic knowledge The proportion of sickness: 0.0051% Susceptible people: no special people Mode of infection: non-infectious Complications: upper gastrointestinal bleeding, ascites, hyponatremia, hepatic encephalopathy
Cause
Purpura - clubbing - cause of hepatic syndrome
Cause:
May cause abnormalities in the liver to cause pulmonary artery shunt, causing purpura and clubbing; dye dilution test can indirectly suggest pulmonary arteriovenous shunt, and indicates that liver cirrhosis is serious, may also be a liver and lung syndrome Performance.
Prevention
Purpura - clubbing - prevention of liver disease syndrome
The disease is mainly caused by cirrhosis, and prevention of the occurrence of cirrhosis can prevent the disease.
1. Adjust daily life and workload, and regularly carry out activities and exercise to avoid fatigue.
2. Maintain emotional stability and avoid emotional excitement and tension. 3. Keep the stool smooth, avoid using stools, eat more fruits and high-fiber foods.Complication
Purpura - clubbing - complications of hepatic syndrome Complications upper gastrointestinal bleeding ascites hyponatremia hepatic encephalopathy
Liver cirrhosis can be complicated by electrolyte imbalance, upper gastrointestinal bleeding, ascites, hyponatremia, hepatic encephalopathy.
Symptom
Purpura - clubbing - symptoms of hepatic syndrome common symptoms clubbing (toe) purpura dyspnea systemic skin and mucous membranes... 5th short bending
Patients with cirrhosis have purpura and clubbing, which can be diagnosed if they can eliminate cardiopulmonary disease. Both men and women can be seen at any age, and children are more common. With the symptoms and signs of cirrhosis, purpura may be accompanied by difficulty breathing, and then the clubbing.
Examine
Purpura - clubbing - examination of liver disease syndrome
First, the medical history: medical history is very important to identify the cause of cyanosis, especially the way and age of the patient's onset. Acute systemic cyanosis with disturbance of consciousness but not obvious dyspnea should pay attention to chemical cyanosis; children or infirm people should suddenly pay attention to intestinal cyanosis after eating brains or kimchi, and should pay attention to intestinal cyanosis. Imagine the possibility of misuse of nitrite; the cyanosis that occurs with the menstrual cycle is characteristic of idiopathic paroxysmal methemoglobinemia.
Second, physical examination: mainly to understand whether the patient has signs of heart, lung or thoracic disease only thoracic deformity, lung voice, heart murmur, etc.) and blood circulation of the hair.
Diagnosis
Diagnosis and differentiation of purpura-caries-hepatic syndrome
Patients with cirrhosis have purpura and clubbing, which can be diagnosed if they can eliminate cardiopulmonary disease.
Differential diagnosis:
1. Edema with difficulty in breathing, purpura: when there is excessive fluid accumulation in the interstitial space of the human body, it is called edema. Purple sable refers to the darkness of the lips and nails. Edema with dyspnea and cyanosis is prompted by heart disease, beriberi, and superior vena cava obstruction syndrome.
2. Purpura and blood hypoxia: Fallot tetralogy is a common congenital heart vascular malformation, ranking first in purpuric congenital heart disease. The most common major clinical symptoms of tetralogy are purpura and blood hypoxia. The time and severity of presentation of clinical symptoms depends on the extent of right ventricular outflow obstruction and the amount of pulmonary circulation blood flow.
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