Pediatric Cast Syndrome

Introduction to Pediatric Plaster Syndrome Gypsumsyndrome, first proposed by Willett in 1878 to describe a case of acute gastric dilatation with nausea and recurrent vomiting after the use of hip herringbone gypsum, was reported in 1983 by Shen Xia in China. In 1987, Yang Quancheng reported 3 cases, including 1 case of 6-year-old girl. The congenital dislocation of the left hip was performed after the Chairi operation. The hip-shaped gypsum was fixed. On the 2nd day, the crying was serious, nausea, abdominal distension was more dramatic, and the bowel sounds were obvious. Weakened, vomited 2 times stomach contents, dehydrated appearance, partial removal of gypsum by liquid therapy, gastrointestinal symptoms disappeared after abdominal gypsum window opening, Evarts believes that the name of gypsum syndrome is actually a misnomer Because it can also occur in different processes for the treatment of severe scoliosis or kyphosis, such as pelvic traction, intraspinal distractor fixation, body wedge gypsum orthopedics, skull-pelvic traction, etc., still using Willett The name of the proposed plaster syndrome. basic knowledge The proportion of illness: 0.002% Susceptible people: children Mode of infection: non-infectious Complications: dehydration hypokalemia

Causes of pediatric plaster syndrome

(1) Causes of the disease

Using various methods of malformation treatment, the superior mesenteric artery is pressed against the transverse part of the duodenum, causing mechanical obstruction to cause the disease. Others such as supine on the brake bed, fixed body plaster, spinal traction and decreased abdominal muscle tone, etc. As a predisposing factor.

(two) pathogenesis

The superior mesenteric artery originates from the abdominal aorta and passes through the lower edge of the pancreatic neck. It crosses the transverse part of the duodenum to the root of the small mesentery at the level of the first lumbar vertebra. The angle of intersection with the abdominal aorta is acute, with an average of 41°. The duodenum and the transverse mesenteric root are connected to the left side of the second lumbar vertebrae and are connected to the empty body. Here, the intestinal tract is often suspended and fixed by the ligament of the smooth muscle fiber bundle from the iliac crest. The most fixed part of the small intestine, the transverse part of the duodenum is completely fixed to the posterior abdominal wall, the superior mesenteric artery in front, the abdominal aorta and the spine in the back, due to the above anatomical features, the duodenum transverse Easy to be blocked by pressure.

Pediatric plaster syndrome prevention

Improve the awareness and vigilance of this disease, improve the orthopedic treatment design and operation technology, so as to effectively prevent the occurrence of this disease.

Pediatric plaster syndrome complications Complications dehydration hypokalemia

Can be hydropower balance disorder, dehydration, alkalosis, hypokalemia and so on.

Symptoms of gypsum syndrome in children Common symptoms Low blood potassium bloating acute gastric dilatation dehydration nausea shock

The gypsum syndrome is caused by the mesenteric artery compressing the duodenum to cause mechanical obstruction. If it is not recognized or delayed, it will lead to acute gastric dilatation, vomiting, hypokalemia, hypovolemia, and alkali poisoning. death.

Examination of pediatric plaster syndrome

Pay attention to blood pH, changes in blood sodium, potassium, chlorine, and calcium.

Should be done X-ray, ECG, abdominal B-ultrasound, etc., ECG can have low potassium performance, visible gastric dilatation and so on.

Diagnosis and diagnosis of gypsum syndrome in children

diagnosis

Diagnosis based on medical history and clinical manifestations.

Differential diagnosis

Identification with acute gastroenteritis, medical history and laboratory tests can help diagnose.

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