Acute cellulitis of the vulva


Introduction to vulvar acute cellulitis Vulvar acute cellulitis is an acute, suppurative, diffuse inflammation that occurs after pathogenic bacteria such as hemolytic streptococcus or staphylococcus invade the vulva or deep loose connective tissue. It spreads rapidly around the disease and has no obvious boundary with normal tissues. This disease can sometimes be caused by anaerobic infection or chemical stimulation. basic knowledge The proportion of illness: 0.005% Susceptible people: women Mode of infection: non-infectious Complications: urethritis vaginitis


The cause of vulvar acute cellulitis

(1) Causes of the disease

Inflammation of the vulva acute cellulitis is caused by damage to the skin or soft tissue: the bacteria invade from the damaged skin, and may also spread from the local purulent lesion to the surrounding area or from the lymph and blood. The pathogenic bacteria are mainly hemolytic streptococcus. A small number of Staphylococcus aureus can also be caused by anaerobic or spoilage bacteria, chemical stimulation (such as improper drug injection) or secondary infection of foreign bodies can also lead to the disease.

(two) pathogenesis

The main pathological changes of acute cellulitis are extensive acute suppurative inflammatory changes in the skin and subcutaneous tissues. There are a large number of neutrophils and lymphocytes infiltrating, blood vessels and lymphatic vessels in the lesions are dilated, and thrombosis, hair follicles, and sebaceous glands are sometimes seen. The sweat glands are all destroyed, and the granuloma can form in the late stage of the lesion. The central area of the lesion is swollen due to inflammation, the subcutaneous tension is increased, and the blood circulation disorder can cause the skin to be purulent and necrotic, and severe cases can cause sepsis.


Vulvar acute cellulitis prevention

Attention to personal hygiene, active treatment of small skin infections and other parts of the infection, pay attention to strict aseptic technique in the injection operation, open injury should be actively treated to prevent the occurrence of this disease.


Complications of vulvar acute cellulitis Complications urethritis vaginitis

Concurrent urethra, vaginal inflammation, blood routine examination of white blood cell count, vaginal discharge examination.


Vulvar acute cellulitis symptoms Common symptoms Painful ulcers and exudation... Cold chills, high fever, crusting, severe pain, hardening, sputum, leukocytosis

Vulvar acute cellulitis is characterized by local red diffuse invasive plaques, unclear boundaries, unrestricted lesions, rapid spread, no obvious boundaries with normal tissues, local red, swollen, hot, severe pain, redness There is a solid feeling on the skin, the depression of the pressure, severe blistering on the surface or dark red ischemic necrosis due to swelling, often with chills, fever, peripheral lymphadenopathy, leukocytosis and other systemic symptoms, and even sepsis Afterwards, the tissue gradually softens and fluctuates. After the ulceration, the pus is formed and ulcers form. After 2 weeks, the chronic cellulitis often appears to be plate-like hardening, flushing, burning and pigmentation, and the pain is not obvious. Hardening atrophy changes similar to scleroderma, also known as indurated cellulitis, if it is deep in the central part of the disease, local redness is not obvious, only local edema and deep tenderness, and the pain is light, compression local, sputum Pronunciation, but at this time the condition is more serious, with high fever, chills, headache, general malaise, increased white blood cell count, cellulite and tendons When the membrane has progressive necrosis, the pus has a foul odor.


Examination of vulvar acute cellulitis

The pus is used for bacterial culture and drug susceptibility testing for the diagnosis and treatment.


Diagnosis and differentiation of vulvar acute cellulitis

According to the patient's local diffuse redness, swelling, heat, pain, unclear boundary, central softening, pus and other clinical manifestations after rupture, can be diagnosed by individual gas-producing bacteria (such as Escherichia coli, anaerobic bacilli, anaerobic) Caused by streptococci, etc., subcutaneous may have a burst of sensation, and some may be accompanied by chills, fever, headache, fatigue, peripheral lymphadenopathy and other systemic symptoms, blood routine examination can be found leukocytosis, neutrophils .

Should be identified with erysipelas, erysipelas is a clear edematous erythema, the lesion is shallow, not purulent.

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