Facial atrophy

Introduction

Introduction to facial lateral atrophy Facial hemiatrophy (facialhemiatrophy), also known as Romberg disease and progressive facial half atrophy, is a rare progressive atrophy of skin, subcutaneous tissue and facial muscle development disorders. basic knowledge The proportion of illness: 0.003% Susceptible people: no special people Mode of infection: non-infectious Complications: epilepsy migraine trigeminal neuralgia

Cause

Facial lateral atrophy

(1) Causes of the disease

The cause is still unclear, and may be related to trigeminal nerve, cervical sympathetic dysfunction leading to vascular movement, nutritional dysfunction, or endocrine dysfunction, or related to craniocerebral, neck trauma, infection or fetal damage.

(two) pathogenesis

The pathogenesis is still not very clear, and may be related to vascular motility and nutritional dysfunction caused by trigeminal nerve and cervical sympathetic dysfunction.

Prevention

Facial lateral atrophy prevention

Early detection and early diagnosis and early treatment.

Complication

Facial lateral atrophy complications Complications, epilepsy, migraine, trigeminal neuralgia

Only some patients were accompanied by seizures or migraine, but about half of the patients had paroxysmal activity recorded on the EEG. Patients with epilepsy, migraine, trigeminal neuralgia, ocular inflammation should be treated accordingly. In severe cases, plastic surgery is feasible.

Symptom

Symptoms of facial side atrophy Common symptoms Semi-lateral dysplasia Neuralgia Skin pigmentation with bronze unilateral atrophy Skin pale blood disorder Obstruction of the frontal bone

On the cheeks of the face, the forehead and the lower jaw begin to have irregular pigmentation or hypopigmentation, or occasionally see local hair whitening, tendon or neuralgia, and later atrophy, progressive development, local skin, subcutaneous tissue, muscle, The tongue and even the bones atrophy successively, the skin appears pigmentation, mane or sebaceous glands, sweat reduces or disappears, severe facial deformity is deformed, thin and depressed, skin is thin, can see the capillaries, often limited to a certain branch of the trigeminal nerve The distribution area gradually affects the side of the whole face, and the midline of the face is obviously contrasted with the healthy side. The demarcation is clearly defined, and sometimes the lateral body atrophy occurs, and the contralateral body cross atrophy, local nerve function or blood supply disorder When the skin temperature is low, pale, spicy and so on.

Examine

Examination of facial lateral atrophy

Histopathology: epidermal atrophy and thinning, basal basophilic degeneration of dermal connective tissue, collagen fiber homogenization, reduction of elastic fibers, sweat glands, sebaceous glands, hair follicles, atrophy, reduced blood vessels, subcutaneous fat disappeared.

Diagnosis

Diagnosis and differentiation of facial lateral atrophy

According to the clinical manifestations, the characteristics of skin lesions and histopathological features can be diagnosed.

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