Ginger fascioliasis

Introduction to ginger schistosomiasis Ginger worm disease (fasciolopsiasis) is a zoonotic parasitic disease caused by fasciolopsis buski, referred to as ginger worm, parasitic in human and pig intestines. Clinically, gastrointestinal symptoms such as abdominal pain and diarrhea are The main, adult worm body is large, the suction cup is developed, and the adsorption force is strong. The mechanical damage of the intestine is obvious compared with other intestinal trematodes. When the number is large, it can cover the intestinal wall, hinder absorption and digestion, and its metabolites can be induced to be metamorphosed after being absorbed. reaction. The adsorbed mucous membrane can cause inflammation, hemorrhage, edema, necrosis, shedding and ulceration, infiltration of neutrophils, lymphocytes and eosinophils in the lesion, increased secretion of intestinal mucosa, frequent abdominal pain and diarrhea, malnutrition Digestive dysfunction, albumin reduction, lack of various vitamins; there may be alternating diarrhea and constipation, and even intestinal obstruction, severe infection in children may have weight loss, anemia, edema, ascites, mental retardation, developmental disorders and so on. In cases of repeated infections, a small number may die from exhaustion and collapse. basic knowledge The proportion of illness: 0.002% Susceptible people: no specific population Mode of transmission: insect vector transmission Complications: intestinal obstruction

Causes of ginger schistosomiasis

Cause of the disease (80%)

People are infected with raw foods accompanied by aquatic foods of the genus Ginger, and patients often have abdominal pain, diarrhea, indigestion, intestinal dysfunction and malabsorption.

Pathogenesis (10%)

Ginger worm has a strong suction cup and strong adsorption capacity, which can cause inflammation of the adsorbed small intestinal mucosa and nearby tissues, punctate hemorrhage, edema and ulcer abscess. The adsorption site often has a large number of neutrophils, lymphocytes and eosinophils. Infiltration of granulocytes, mucous membrane epithelial cells secrete a large amount of mucus, and the lesions may have hemorrhage in the intestinal wall. The worms attach to the intestinal wall of the host, ingest intestinal nutrients, and cover the mucosa of the intestinal wall, hindering the absorption and digestion of the intestinal tract. Intestinal dysfunction and malnutrition, metabolites and secretions of the worm can cause host morbidity and eosinophilia. The severity of the lesion is related to the number of insects in the parasite host, usually several to dozens. In severe cases, there are hundreds or even thousands of worms, and most of the worms can block the intestinal wall to form intestinal obstruction.

Ginger disease prevention

In order to prevent the spread of this disease, we must first strengthen health publicity and education, eat cooked water stalks, sputum, etc., or rinse with water before eating and use a knife to cut off its husk. At the same time, we must do a good job of manure management. Harmless manure should not be used as a fertilizer for planting aquatic plant ponds and paddy fields. Pigs should also be properly treated to prevent pigs from being infected; it is forbidden to place pigs on the edge of the pond where aquatic plants are grown.

Ginger worm complications Complications, intestinal obstruction

Allergies, intestinal obstruction, granulocyte infiltration, lung, intestinal bacterial infections, etc.

Symptoms of ginger schistosomiasis common symptoms abdominal pain diarrhea nausea appetite lack of molars bowel twitching flatulence gas pain edema

The incubation period is 1 to 3 months. The symptoms of mild infection are mild or asymptomatic. In moderate to severe cases, there may be anorexia, abdominal pain, intermittent diarrhea (mostly dyspepsia), gastrointestinal symptoms such as nausea and vomiting. Located in the upper abdomen and the right quarter of the ribs, a small number in the umbilical week, occurred in the morning fasting or after meals, mainly abdominal pain, occasionally severe pain and cramps, patients often have bowel sounds hyperthyroidism, intestinal peristalsis, intestinal flatulence, Many patients have a history of automatic worms or worms. Children often have neurological symptoms such as poor nighttime sleep, molars, convulsions, etc. A small number of patients may have edema and ascites due to long-term diarrhea, severe malnutrition, and severe exhaustion in patients with advanced disease. Deficiency or secondary lung, intestinal bacterial infection, causing death, occasionally the worms gather into a group leading to intestinal obstruction.

Anyone with a history of raw aquatic plants and varying degrees of gastrointestinal symptoms should be considered in the epidemic area of ginger worm disease. The diagnosis depends on the detection of ginger worm eggs in the feces and 3 smears of one stool. More positive results, less eggs can be concentrated by formaldehyde-ether method to increase the detection rate.

Ginger worm disease check

Various egg-concentration methods can improve the detection rate, but the diagnosis of ginger schistosomiasis needs to consider the number of parasitic insects and clinical manifestations. The former can be roughly impressed by the counting method, and the patient's red blood cell count and hemoglobin often decrease slightly. The white blood cell count is slightly increased, eosinophils can be increased to 10% to 20%, even up to 40%, fecal occult blood test is positive, and the feces can be found by direct smear method or sediment collection method. The former is easy to miss the diagnosis of mild infection, the latter can increase the detection rate, and can also use the quantitative transparent method (ie modified Kato smear method), which can be qualitative and can be used for egg counting.

X-ray examination showed slow bone growth or dwarfism.

Diagnosis and identification of ginger schistosomiasis

diagnosis

Diagnosis can be based on medical history, clinical symptoms, and laboratory tests.

Differential diagnosis

The disease should be distinguished from the tsutsugamushi disease. The ginger worm eggs should be differentiated from the eggs of the larvae of the liver and the eggs of the larvae.

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