Chest wall phlebitis

Introduction to chest wall phlebitis Pectoral wall phlebitis is more common in middle-aged women. It generally has no serious clinical symptoms. It has pain in the lateral chest wall and axillary fossa. It can touch a cord through the lateral chest wall to the armpit, and can extend to the upper part of the umbilicus. The surrounding skin can be presented. Congestive erythema, sometimes accompanied by edema. It gradually subsided, the congestion was replaced by hyperpigmentation, and the erythema turned brown. A small number of patients can cause reactions, such as chills, fever, white blood cells, etc., patients often complain of pain and swelling. basic knowledge The proportion of the disease: the incidence rate of people over 50 years old is about 0.004% - 0.006% Susceptible people: more common middle-aged women Mode of infection: non-infectious Complications: thrombophlebitis

Causes of chest wall phlebitis

Causes

It is also caused by Mand's disease due to thrombophlebitis and peri-venous inflammation of the venous wall of the chest wall and the thoracic and abdominal wall.

Chest wall phlebitis prevention

Increase the amount of tissue perfusion, reduce clinical symptoms, in order to increase the amount of tissue perfusion to provide sufficient tissue metabolism of oxygen and nutrients, the nursing measures mainly from the establishment of collateral circulation, avoid vasospasm and protect the affected limbs Start.

Chest wall phlebitis complications Complications thrombophlebitis

May cause pain in the chest.

Common symptoms of chest wall vein venous axillary pain chest wall pain venous blood flow slow inflammatory cell infiltration venous thrombosis

More common in middle-aged women, generally no serious clinical symptoms, pain in the lateral chest wall and axillary fossa, a cord can be reached through the lateral chest wall to the armpit, and can be extended downwards above the umbilicus.

Examination of chest wall phlebitis

1, venous pressure measurement: increased venous pressure of the affected limb, suggesting that the proximal end of the vein is blocked at the lateral pressure;

2, ultrasound: two-dimensional ultrasound imaging can be directly improved to see the conclusion of thrombosis in the large vein, with Doppler to measure the experience of intravenous blood flow velocity, and observe whether the normal leadership response of breathing and compression action exists, such inspection is near The diagnostic rate of deep venous thrombosis in the next month can reach 95%; the sensitivity to the diagnosis of distal patients is only 50%-70%, but the specificity can reach 95%;

3, radionuclide health check: 125I fibrinogen scan is used for the diagnosis of children with this disease, contrary to ultrasound examination, the detection rate of deep vein thrombosis in the gastrocnemius can be as high as 90%, and the proximal end The diagnosis of deep vein thrombosis has been poorly specific. The main disadvantage of this test is that it takes 48-72 hours to inject the radionuclide to show the effect.

4. Impedance plethysmography (IPG) and venous flow spirometry (PRG): The former uses skin electrodes, the latter uses a pneumatic cuff to measure changes in venous volume under physiologically changing conditions, with venous obstruction, with breathing and cuffs The volume of the volume wave caused by deflation and deflation is small. The positive rate of the diagnosis of the proximal deep vein snow mountain formation by the poor test is up to 90%, and the sensitivity to the diagnosis of the distal side is significantly reduced.

5, deep venography

The contrast agent is injected from the superficial vein of the foot, and the cuff is used at the proximal end. It is easy to start the contrast agent directly into the deep venous system. If there is a venous filling defect, the diagnosis and positioning diagnosis can be made.

The diagnosis of superficial venous thrombosis is easier, and the signs of local discharge symptoms are more obvious.

Diagnosis and diagnosis of chest wall phlebitis

According to the clinical manifestations, combined with pathological examination, there is inflammatory cell infiltration of the venous wall, but the lumen is not blocked and can be diagnosed.

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