Foramen magnum meningioma

Introduction to occipital macroporous meningioma Occipital macroporous meningioma, also known as meningioma in the large occipital region, refers to meningiomas that occur around the occipital foramen. Half of them occur in the forefront of the foramen magnum, which can grow into the brain or grow into the cervical canal. Often caused by the compression of the medulla. basic knowledge The proportion of illness: 0.003% Susceptible people: no specific population Mode of infection: non-infectious Complications: bulbar paralysis

Causes of occipital macroporous meningiomas

Causes:

No relevant information.

Pathogenesis:

The cranial spinal cord type originates from the ventral or ventral lateral dura mater of the brainstem. The basal is located on the lower slope, above the occipital macropores, and grows toward the occipital foramen. The spinal cord cranial shape grows from the cervical segmental meningioma to the skull, and the blood supply is mainly Branches from the vertebral artery, occipital artery, and pharyngeal artery.

Prevention of meningioma of the occipital foramen

The first is diet, followed by prevention.

Complications of occipital foramen meningiomas Complications, medullary paralysis

In the surgical treatment, if the injury is IX, X brain nerve and medulla oblongata, medullary palsy may occur, and the injury of the XI brain nerve may cause one side of the shoulder to sag.

Occipital macroporous meningioma symptoms Common symptoms Muscle atrophy, increased intracranial pressure, hydrocephalus

The disease develops slowly. The average course of treatment in the hospital is 2.5 years. The early manifestation is pain in one side of the neck. With the development of the disease, limb numbness gradually appears after several months. The upper limbs are more common, followed by the lower limbs, tumor compression and medulla and high neck. In the marrow, the patient may have weakness in both upper limbs. In severe cases, limb muscle atrophy may occur, and tendon reflexes may be weakened. When the cerebellum is involved, the patient may have unstable walking and joint movement disorder.

Neurological examination revealed pain or temperature sensation diminished or disappeared. One-four of the patients showed clinical manifestations similar to syringomyelia. Cranial nerve damage was common with lesions of the X and XI cranial nerves, and the damage of the X-brain nerves and the brainstem The down-sensing conduction beam is related to pressure. When there is only XI brain damage in the clinic and there is no X-brain nerve damage, it indicates that the tumor position is low, and the tumor of the jugular foramen can be excluded. When the tumor is compressed, the obstructive hydrocephalus is formed. At the time, the patient developed symptoms of increased intracranial pressure, such as headache, vomiting, and optic disc edema.

Examination of large occipital meningiomas

In the 1970s, the diagnosis mainly relied on spinal iodine oil and ventricle lipiodol angiography. The contrast picture showed clear filling defects in the occipital macropore area. In recent years, foreign countries have used CT scan of the genus Glucosamine myelography instead of cumbersome traditional myelography. Cerebral ventricular iodine angiography, 3/4 cases were diagnosed by contrast CT scan. MRI is the best method for diagnosing posterior fossa and upper neck tumors. With enhanced MRI scan, almost 100% of occipital macropore tumors can be diagnosed. .

Diagnosis and diagnosis of meningioma of the occipital foramen

According to the medical history, clinical manifestations, combined with CT and MRI imaging examination, can generally be diagnosed.

The clinical development of occipital macroporous meningioma is slow, and needs to be differentiated from high cervical medullary cavity, ring pillow deformity, cervical spondylosis, multiple sclerosis, high cervical intramedullary tumor, etc., but the differential diagnosis is not confirmed by CT or MRI. difficult,

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