Schizophrenia

Introduction to schizophrenia During the embryonic period, due to the malformation of the spinal cord or spinal canal, the spinal cord is split into two halves, called schizophrenia. This disease is extremely rare, and the patient may have no obvious clinical symptoms, and some may have spinal cord splitting syndrome. basic knowledge The proportion of illness: 0.001% Susceptible people: no specific population Mode of infection: non-infectious Complications: scoliosis Spinal tuberculosis dysplasia Cerebellar tonsil sacral recessive spina bifida Dysphagia

Cause of schizophrenia

Congenital developmental defects in the spinal cord (45%):

In the early stage of the embryo, the central tube is closed abnormally. If the left and right side of the neural crest are bent forward before the contact, the first contact with the bottom plate will form two neural tubes, which will later develop into schizophrenia, also known as double Spinal cord disease.

Intraspinal bone dysplasia (35%):

In the development of the spinal canal, due to the abnormal proliferation of bone, it grows into the central part of the spinal cord, and the spinal cord is divided into left and right halves, forming a non-full-length schizophrenia of the spinal cord, which is partial schizophrenia.

Pathogenesis

According to the pathological manifestations of mitosis, there are two manifestations: one is the two halves of the spinal cord divided into two, each with a dural sheath, in which the bone is separated from the cartilage tissue; the other is in a dural sheath Inside, contains two half of the spinal cord, which is separated by fibrous tissue septum. The two half of the spinal cord can each contain a central tube, and some are half thick and half thin. This kind of total fissure is rare, and there are more splits in the spinal cord. It can be seen in the lower thoracic and lumbar segments, which can cause tethered cord syndrome.

Spinal schizophrenia prevention

1. Stay optimistic and happy

Stronger long-term or repeated emotional stress, anxiety, irritability, pessimism and other emotional changes can make the balance of cerebral cortex excitability and inhibition process imbalance, so that muscle jump is aggravated, and muscle atrophy develops.

2. Pay attention to prevent colds and infections

Patients with syringomyelia muscle atrophy have low autoimmune function or some kind of immunodeficiency. Once they have a cold, their condition is aggravated, the course of disease is prolonged, muscle weakness is weak, and muscle spasm is aggravated. Especially patients with bulbar paralysis are prone to pulmonary infection, if not Timely prevention and treatment, poor prognosis, and even endanger the lives of patients. Gastroenteritis can lead to dysfunction of intestinal bacteria. In particular, viral gastroenteritis has different degrees of damage to the anterior horn cells of the spinal cord, which causes the patient to gain muscle extension, decrease muscle strength, and repeat or aggravate the condition.

3. Reasonable diet and maintain normal digestive function.

Complications of schizophrenia Complications, scoliosis, spinal tuberculosis, stenosis, cerebellar tonsillar, inferior sacral spine, dysphagia

Syringomyelia often has scoliosis, cerebellar tonsil, sacral deformity, recessive spina bifida, neck occipital deformity, arched foot and other diseases.

After the development of syringomyelia lesions and pyramidal and extrapyramidal bundles, the lower extremities gradually developed spastic paralysis, and the lower limbs were positive for pyramidal tract. When one cervical spinal cord was damaged, the descending sympathetic fibers were destroyed. There may be Hornor syndrome, a more common autonomic disorder, skin dystrophies such as keratosis, reduced hair, and vasomotor disorders.

In the later stage of syringomyelia, the cavity often involves the facial onion skin-like pain and sensation of the facial trigeminal nucleus, which develops from the lateral to the nasolabial; the vestibular cerebellar pathway is affected by dizziness, nystagmus and gait stability; involving the hypoglossal nerve Nuclear extensor muscles and fasciculation; involving the suspected nucleus causes difficulty in swallowing drinking water and cough; involving facial nerve nucleus with peripheral facial paralysis and other symptoms.

Symptoms of schizophrenia Common symptoms Lower extremity muscle, cervical and cervical epidural lesions, spinal hemisection syndrome, high paraplegia

Spinal schizophrenia can be clinically asymptomatic, but some patients have similar manifestations of tethered cord syndrome, including lower extremity sensation, dyskinesia and pain, and severe lower extremity spasm and dysfunction.

Examination of schizophrenia

X-ray plain film and CT scan are more difficult to display, and MRI scan can clearly show schizophrenia, as well as osteophytes or spurs in between.

Diagnosis and differentiation of schizophrenia

Spinal schizophrenia is rarely able to make a correct diagnosis from clinical manifestations before surgery, and MRI scans help to confirm the diagnosis.

The disease should be differentiated from the spine fissure. Patients with spondylolisthesis may have local skin hairy, purple spots, pits, pigmentation and other clinical manifestations, and symptoms of nerve damage, especially long-term enuresis or obvious urinary incontinence, should be considered as Caused by spina bifida, spinal X-ray film and CT and MRI scans help to differentiate the disease.

The material in this site is intended to be of general informational use and is not intended to constitute medical advice, probable diagnosis, or recommended treatments.