Throat burns and chemical injuries

Introduction to throat burns and chemical injuries Throat burns and chemical injuries due to inhalation of high-heat steam, accidental drinking of boiling water, head and neck burns due to inhalation of flame or dry hot air caused by mucous membrane burns, misuse of strong acid, strong alkali, inhaled ammonia, cyanide, chlorine, iodine, bromine, phosphorus and other high concentrations Aerosols and chemical gases can directly burn the mucous membranes of the throat and lower respiratory tract and cause systemic poisoning. Burns are in the mouth, and the face and the sputum will be the most severely wrinkled outside. The vapour and mist damage will be obvious in the area of the epiglottis and the glottic area. The chemical and corrosive injuries are lighter in the mouth and throat, and the esophagus is heavier. Immediately after injury, pulmonary edema may occur in lower respiratory tract injury, aggravating dyspnea with cough, coughing up bloody secretions, severe symptoms may cause systemic poisoning symptoms such as lethargy, dehydration, high fever, shock, etc. Renal failure, liver damage caused by death, throat, oral mucosa congestion, swelling, erosion, ulcers and leukorrhea formation, severe vocal cord redness, pseudomembrane visible under the glottis as signs of difficulty breathing. basic knowledge Sickness ratio: 0.05% Susceptible people: more common in young children caused by eating Mode of infection: non-infectious Complications: dehydration shock

Throat burn and chemical injury

Ingestion of boiling water or chemical corrosive agents can cause damage to the oral mucosa and pharyngeal mucosa. In severe cases, there are severe symptoms of systemic poisoning. Scalding is more common in young children.

Throat burn and chemical injury prevention

The most important thing is to prevent and treat dyspnea, to give oxygen and intravenous drops of hormones, to reduce laryngeal and pulmonary edema, and to prevent scarring.

Throat burn and chemical injury complications Complications dehydration shock

Severe patients may have symptoms of systemic poisoning, such as lethargy, dehydration, high fever, shock, etc., and even cause renal failure, liver damage and death.

Throat burns and chemical injury symptoms Common symptoms Difficulty breathing High heat congestion Dehydration Drowsiness Tissue necrosis shock

Burns are in the mouth, and the face and the sputum will be the most severely wrinkled outside. The vapour and mist damage will be obvious in the area of the epiglottis and the glottic area. The chemical and corrosive injuries are lighter in the mouth and throat, and the esophagus is heavier.

Immediately after injury, pulmonary edema may occur in lower respiratory tract injury, aggravating dyspnea with cough, coughing up bloody secretions, severe symptoms may cause systemic poisoning symptoms such as lethargy, dehydration, high fever, shock, etc. Renal failure, liver damage caused by death, throat, oral mucosa congestion, swelling, erosion, ulcers and leukorrhea formation, severe vocal cord redness, pseudomembrane visible under the glottis as signs of difficulty breathing.

The damage of the high heat vapor to the mucous membrane is heavier than that of the dry hot gas. When the steam encounters the mucous membrane, it agglomerates and releases the latent heat to damage the mucous membrane. The ammonia mist, phosphorus, iodine, chlorine and the like combine with the water in the mucous membrane to produce different acid salts and burn the mucous membrane. Mostly sodium hydroxide, with strong water absorption, fat saponification and protein solubilization, so the lesions often infiltrate into adjacent tissue development, strong acid usually causes mucosal dry coagulative necrosis, mostly limited to the parts that have been in contact with corrosive agents for a long time.

At the beginning of the lesion, mucosal congestion, edema, erosion, and cellulose exudation formed a white membrane. Mucosal edema began at 1-2 h after injury, peaked at 4-8 h, and edema gradually subsided after 2-3 d. Unequal ulcers, severe local tissue necrosis, and even perforation of the esophagus or trachea, about 10d after the ulcer in the formation of granulation tissue, tissue fibrosis, and gradually formed scars, 4-6 weeks gradually appear scar adhesion and stenosis.

Throat burn and chemical injury check

It can be seen that the soft palate, the uvula, the posterior pharyngeal wall, and the mucous membrane of the epiglottis are blistering, erosive, or covered with a white membrane.

Diagnosis of throat burn and chemical injury

Distinguish from the injury of the throat, the burn is in the mouth, the face and the sputum will be the most severe outside the vulture, the vapor and fog damage will be obvious in the epiglottis and the subglottic area, and the oral and pharyngeal damage will be lighter in the chemical corrosive injury. weight.

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