Traumatic subdural effusion

Introduction to traumatic subdural effusion Traumatic subdural effusion refers to the formation of a large amount of fluid retention in the subdural space after trauma, also known as traumatic subdural hydroma. Due to brain injury, the brain tissue moves strongly in the cranial cavity, causing the arachnoid membrane to be torn, and the cerebrospinal fluid flows through the hiatus to the subdural space between the subdural and the arachnoid. The incidence is craniocerebral injury. 1.16%, accounting for about 10% of traumatic intracranial hematoma. basic knowledge The proportion of illness: 0.001% Susceptible people: no special people Mode of infection: non-infectious Complications: cerebral palsy, coma

Traumatic subdural effusion

The mechanism of traumatic subdural effusion is that the arachnoid rupture is like a one-way valve. The cerebrospinal fluid can continuously flow out with the patient's struggle, suffocation, coughing, etc., but can not return to the subarachnoid space. The formation of hydrosarcoma-like effusions under the sub-mening causes the consequences of local brain compression and progressive intracranial pressure.

Traumatic subdural effusion prevention

1. The disease is a traumatic disease, pay attention to safety, drive carefully, keep the home environment bright, walk carefully, watch children and avoid trauma.

2. In patients with subdural effusion, primary brain injury is generally mild. If the treatment is timely and reasonable, the effect is good. If the primary brain injury is severe and/or accompanied by intracranial hematoma, the prognosis is poor. The rate can reach 9.7% to 12.5%.

Traumatic subdural effusion complications Complications, cerebral palsy

The disease is easy to form due to intracranial hypertension and cerebral palsy, resulting in sudden coma, resulting in death of heartbeat breathing.

Traumatic subdural effusion symptoms common symptoms headache and vomiting intracranial pressure increased papilledema

The clinical manifestations of subdural effusion resemble subdural hematoma. There are also acute, subacute and chronic points, which are difficult to distinguish before surgery. The clinical features are light or medium closed head injuries, and the primary brain damage is often light. After the injury, there is a progressive increase in headache, vomiting and papilledema and other manifestations of increased intracranial pressure. The course of the disease is mostly subacute or chronic. Occasionally, it can be an acute process. In severe cases, it can also cause the sacral leaf to return to the sputum. 30.4% of patients had unilateral dilated pupils, about half of them were consciously progressively degraded and positive for pyramidal tract signs. The amount of subdural effusion was generally 50-60 ml, and many of them were up to 150 ml. Their traits were mostly bloody. Cerebrospinal fluid, turned to yellow clear liquid for a long time, the protein content is slightly higher than normal.

Traumatic subdural effusion examination

The diagnosis of this disease must rely on special examinations, such as CT or MRI. Sometimes, even with CT scan, it may be confused with an isodose or low-density subdural hematoma. However, the signal on the MRI image is close to the cerebrospinal fluid. The hematoma signal is strong, especially in the T2-weighted image, the hematoma is high-intensity signal, which can be identified.

Diagnosis and diagnosis of traumatic subdural effusion

CT scan may also be confused with subdural hematoma of equal density or low density, but the signal of effusion on the MRI image is close to the cerebrospinal fluid, and the hematoma signal is strong, especially in the T2-weighted image, the hematoma is high. Signal can be identified.

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