Otogenic hydrocephalus

Introduction

Introduction to otogenic hydrocephalus There are two kinds of otogenic hydrocephalus. One is cerebrospinal fluid, which increases the true hydrocephalus of ventricle dilatation. The other is cerebral venous circulatory cranial hypertension, which is pseudo-hydrocephalus after sinus thrombosis. The latter is more common. After suffering from sinus thrombophlebitis, it causes cerebral venous circulation disorder, which is manifested as increased intracranial pressure. It is characterized by cerebral venous swelling and tissue edema. The brain pressure is high but the ventricle is dilated. It is different from the hydrocephalus with general cerebrospinal fluid and dilated ventricles. It is called otogenic hydrocephalus, also known as otogenic intracranial hypertension. basic knowledge The proportion of illness: 0.02% Susceptible people: no special people Mode of infection: non-infectious Complications: optic atrophy

Cause

Etiology of otogenic hydrocephalus

Lateral sinus thromboembolism 30%):

Lateral sinus thrombophlebitis causes embolization of the right dominant sinus or bilateral sinus, causing cerebral venous edema, cortical punctiform hemorrhage, brain tissue infarction softening, brain pressure swell, but cerebrospinal fluid is not much, the ventricle does not expand, On the contrary, there is compression and small, so it is pseudo hydrocephalus. The rate of side sinus thrombophlebitis was 2.5%.

Obstruction of venous return (30%):

In sigmoid sinus thrombophlebitis, the intracranial venous return is blocked, and the arachnoid granule absorption dysfunction produces traffic hydrocephalus. Meningitis and brain abscess can cause interventricular and median holes. The lateral or midbrain aqueduct is narrowed or blocked, which blocks the circulation of cerebrospinal fluid.

Intracranial infection factors (30%):

Intracranial infection complicated with meningitis and brain abscess, due to arachnoid adhesion or obstruction of the aqueduct, resulting in increased secretion or absorption of cerebrospinal fluid, resulting in increased cerebrospinal fluid, the formation of hydrocephalus.

Prevention

Otogenic hydrocephalus prevention

There are not many reports on otogenic hydrocephalus at home and abroad. For the prevention of etiology, such as brain abscess, when treating brain abscess, prevent this disease to prevent it from happening.

Complication

Otogenic hydrocephalus complications Complications optic atrophy

This disease is often complicated by progressive fundus edema and optic atrophy.

Symptom

Otogenic hydrocephalus symptoms common symptoms otogenic vertigo nausea visual impairment hydrocephalus

Recently suffered from intracranial complications such as lateral sinus thrombosis phlebitis, high lumbar puncture pressure, sometimes fundus edema, no space-occupying signs, persistent headache, nausea, progressive vision loss, or even complete blindness, sometimes accompanied by extraocular Show muscle paralysis and so on.

Examine

Examination of otogenic hydrocephalus

CT and MRI can exclude intracranial space-occupying lesions, and superior sagittal sinus venography shows thrombosis in the lateral sinus.

Diagnosis

Diagnosis and diagnosis of otogenic hydrocephalus

The clinical diagnosis of otogenic communication hydrocephalus needs to be differentiated from otogenic meningitis, otogenic brain abscess and otogenic obstructive hydrocephalus. The latter often coexists with brain abscess, showing progressive intracranial pressure. Increased, CT and cerebrospinal fluid examination have abnormal changes, such as not active treatment, can develop into cerebral palsy and death, treatment principles to completely remove the ear lesions, the application of broad-spectrum antibiotics, lowering intracranial pressure (available drugs or lumbar puncture) However, it is necessary to grasp the amount of liquid discharge, so as to avoid the formation of large sacral sacs, severe symptoms, feasible decompression of the diaphragm or ventriculo-peritoneal shunt.

The material in this site is intended to be of general informational use and is not intended to constitute medical advice, probable diagnosis, or recommended treatments.

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