Renal atheroma

Introduction to renal artery atherosclerosis Renal atherosclerosis Renal atherosclerosis A clinical syndrome consists of a rapid or slow decline in renal function based on the amount of obstructed renal arterioles. The incidence of renal embolism increases with age. The most important non-cardiac renal emboli are ruptured aortic plaques. Renal atherosclerosis can occur spontaneously in the case of diffuse aggressive atherosclerosis or secondary to vascular surgery, angioplasty or angiography. Renal atherosclerotic emboli are typically non-swelling, irregular in shape, and more prone to incomplete occlusion with secondary ischemic atrophy rather than renal infarction. The foreign body reaction often occurs immediately, causing intimal hyperplasia, giant cell formation and proliferation, and further vascular stenosis. Pathological reactions may contribute to renal function often progressively 3 to 8 weeks after atherectomy. basic knowledge The proportion of the disease: 0.01%-0.02% (the above is the proportion of patients with kidney disease) Susceptible people: no special people Mode of infection: non-infectious Complications: gastrointestinal fistula gastrointestinal tract with cancer syndrome

Cause of renal atherosclerosis

Cause:

The incidence of renal embolism increases with age. The most important non-cardiac renal emboli are ruptured aortic atherosclerotic plaques (atherosclerotic emboli). Renal atherosclerotic embolism can be used in diffuse aggressive atherosclerosis. Spontaneous or secondary to vascular surgery, angioplasty or angiography during hardening.

Atherosclerotic emboli are typically non-expandable, irregular in shape, more prone to incomplete occlusion with secondary ischemic atrophy rather than renal infarction, often followed by foreign body reactions, causing intimal hyperplasia, giant cell formation And hyperplasia and further vascular stenosis, pathological reactions may contribute to renal function often 3 to 8 weeks after atherectomy, progressive decline.

Renal artery atheroma embolism prevention

1. Control calories: The calorie intake must be balanced with the calories burned. Keep this balance within the standard weight range by properly balancing the diet and strengthening physical activity.

2. Low-fat diet: Eat less animal oil, replace it with vegetable oil, such as soybean oil, peanut oil, corn oil, etc., the dosage is 25 per person per day, less than 750 grams per month, to limit the amount of cholesterol in the food, each Every day should be less than 300 mg, egg yolk and liver, kidney and other animal internal organs have higher fat content, should be less credit.

3. Low-salt diet: patients with atherosclerosis often have high blood pressure, so use a low-salt diet. Do not exceed 5 grams of salt per day. Add salt and mix well after cooking. If you put salt in cooking, cook. The dishes are still very light and difficult to enter. In order to increase appetite, you can add some vinegar, ketchup or sesame sauce when cooking. In addition to seasoning, you can also promote digestion and absorption. The sesame paste contains high calcium and regular credit. It can supplement calcium, calcium ion can increase the density of vascular endothelium, and it has certain benefits to prevent cerebral hemorrhage.

4. Pay attention to drinking water: drink less sugary drinks and drink more natural drinks and water, especially in the morning and evening. The biggest advantage of drinking water is that it can dilute the blood and prevent blood vessel embolism.

5. Appropriate exercise: only pay attention to diet conditioning can not completely prevent the occurrence of atherosclerosis, but also need to exercise properly, to undergo a health check, to determine that the body has no other abnormal phenomena, before you can start walking, walking and other moderate movements Further walks and jogs, and gradually transition to continuous jogging and climbing, swimming and other sports.

Renal atheroma embolism complications Complications, gastrointestinal tract, gastrointestinal tract with cancer syndrome

The most common complication is renal parenchymal necrosis, followed by embolism often affecting other abdominal organs (such as the pancreas, gastrointestinal tract).

Renal artery atherosclerotic symptoms common symptoms hematuria renal arteriosclerosis renal artery obstruction

Renal atherosclerosis should be suspected in patients with idiopathic renal failure over 60 years of age, especially when there is a sign of progressive arteriosclerosis, which is often occult, lacking the clinical features of renal infarction, spontaneously The exact time of atherosclerotic embolization is more difficult to determine than patients undergoing vascular surgery, angioplasty, angiography or anticoagulant therapy (eg warfarin, fibrinolytic drugs).

Diagnosis may be difficult in patients with spontaneous disease or clinical atypical symptoms. In some patients, urine sediment contains hematuria, and a few have red blood cell casts. These manifestations should be suspected of acute glomerulonephritis or vasculitis. Especially if extra-renal manifestation exists, because of the involvement of multiple organs, renal atherosclerosis and polyarteritis may be confused, percutaneous renal biopsy seems to detect about 75% of cases, cholesterol crystals in the embolism when the tissue is fixed It is soluble and appears as a double concave, needle-shaped fissure with pathological features in the occlusion vessel.

Renal artery atherectomy

Most patients with spontaneous renal atherosclerotic embolism have azotemia and manifest as slow progressive renal failure, and those with atherosclerotic thrombosis after vascular surgery can develop acute oliguric renal failure, renal failure patients Usually no kidney-related symptoms, although angiotensin II-mediated hypertension can occur in some cases, urine analysis is often benign microalbuminuria, but occasionally nephropathy range proteinuria, acidophilic in the acute phase Increased cells, eosinophilic urine and transient hypocomplementemia.

Embolization often affects other abdominal organs (such as the pancreas, gastrointestinal tract). The extensive signs of peripheral embolism (such as reticular leukoplakia, painful muscle nodules, and obvious gangrene) strongly suggest diagnosis, but not often, retinal embolization It can lead to sudden blindness, and the orange-yellow spots in the retinal arterioles can be seen during ophthalmoscopy.

Diagnosis and diagnosis of renal atherosclerosis

diagnosis

Diagnosis can be based on medical history, clinical symptoms, and laboratory tests.

Renal atherosclerosis and polyarteritis may be confused, need to be identified, percutaneous renal biopsy can be identified.

The material in this site is intended to be of general informational use and is not intended to constitute medical advice, probable diagnosis, or recommended treatments.